DoctorsToTrust

http://www.DoctorsToTrust.com presents episode 1089 | Dr Nadir Ali Low Carb Denver podcast Age associated w/ mitochondrial dysfunction, IR & lipotoxicity... Study: younger 20s & older 70s -similar in weight, fat mass, lean mass, BMI -Kraft test showed: Older: -higher glucose & insulin -higher muscle & liver fat -40% lower ATP power Statins: lack of data, but inferential information shows... Statins -cause IR -poisons mitochondria by depleting Coq10 -increases liver's & pancreas' VLDL receptors so they become fatty & toxic Damaged mitochondria [yellow] bond with healthy in complementation: to prevent forming free radicals... Or, mitochondria can separate damaged part into: -daughter: damaged -daughter: healthy Damaged daughter cleared from cell by mitophagy In setting of exercise, healthy mitochondria: ATP used as fuel... -AMP goes up, giving strong signal to cell to produce mitochondrial biogenesis factor[PCG-1alpha] Initiates -mitophagy -fusion, fission -biogenesis In setting of sedentary behavior, lipotoxicity, IR... -creating lots of toxic fat damaging mitochondria -won't burn freely -generate lots of free radicals [causing aging] -toxic fat [ceramide] -forms channel leaking out critical proteins -program cell for death -leading to sarcopenia |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbconferences.com Original Youtube: https://youtu.be/4-fQJAifTg0?si=w0wgsIPqM8zxxchu This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1082 | Dr Ken Sikaris Low Carb Down Under podcast Study: 3g carbs; 71% fat -TC & LDL increased "Typical of most on low carb, high fat diet" In this study, participants started with healthy TG -it did not change Why is happening? -VLDL changes to IDL as body takes fat out of it... -then to LDL -both LDL and fragments of IDL left go back to liver In high carb, however, LOTS of VLDL -tissues don't want it -not making IDL or LDL -VLDL remains, crashes around, becomes sdLDL High fat diet [in fat-adapted person]? -VLDL made, body using the fat, so normal process -quickly converted to IDL & LDL -more traffic in this fat cycle, so more LDL in blood -that is why more LDL on high fat diet MOST ON LCHF DIET -higher TC; LDL; HDL -lower TG As shown on these randomized, control trials -AND LOWER sdLDL |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/MXkE8_NdAyQ?si=ce1CsJOGFZGfglIG This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1081 | Dr Ken Sikaris Low Carb Down Under podcast IDL, assumed to not be there, -up to 1.5 of the 6 TC [for this patient] is normally IDL -ignored on standard lipid profile -accounts for large part of TG "this is a game that has been played, that has caused a lot of damage... time to stop" Case 1: patient changed lipid profile with low carb eating... -TC 6.5 to 6.9 -TG 1.6 to 0.7 [taken outside of 1.5 level of concern] -HDL up and LDL up slightly -sdLDL improved from 11% to 1% lowering TG = lowering sdLDL "Don't know why IDL did not change" Case 2: patient -TC 5.2 and LDL 1.5 so doctor: 'no need for statin' -TG 2.1 so 17% of LDL is sdLDL! Onto low carb eating... both HDL & LDL increase [doctors concerned] BUT sdLDL disappeared! "That is bottom line for this patient's cardiovascular risk" |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/MXkE8_NdAyQ?si=ce1CsJOGFZGfglIG This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1081 | Dr Ken Sikaris Low Carb Down Under podcast VLDL, fat-rich particle, crashes into LDL, it's 'child' LDL takes fat from VLDL....leading it to become small, dense -rejected by liver, so stays in blood stream CANNOT MEASURE LDL but predict it from TG -high TG=more sdLDL If TG 2.5 mmol/L [[221 mg/dL] then... MOST OF LDL IS sdLDL LDL, when staying blood changes through -oxidation; glycation -taken up by immune system's macrophages -these are in vessel walls -keep blood clean -when too much, it clogs vessel walls with plaque Pictures of LDL particles -when small, dense they cannot enter liver "small & dense" means slightly smaller than LDL PICTURE: A vs B is actual relative size difference -very subtle change in size will be rejected by liver -26 nanometers vs 24 The presence of sdLDL correlates with all cardiovascular risk markers -blood pressure -weight -insulin & glucose levels Blood lipid test showing all particle sizes and types... for this patient: -smallest is HDL -has low levels small, dense LDL -has LDL, IDL, and VLDL Transformation route is VLDL....IDL....LDL....sdLDL Everyone has IDL, but it is not included in calculating levels This patient is beginning to have sdLDL |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/MXkE8_NdAyQ?si=ce1CsJOGFZGfglIG This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1080 | Dr Ken Sikaris Low Carb Down Under podcast Particles that end of in blood vessels, block them, has NOTHING to do with nature... LDL, instead of returning to liver, can stay in blood and -become smaller, harder -glycated or oxidized Then CANNOT RETURN TO LIVER Rubbish pits collect it [macrophages] -if in blood vessels: TROUBLE Fructose can be converted to triglycerides [TG] in liver... -normally sent out to tissues as VLDL fat packet -if too much and no tissue wants it, it hangs around in blood' 'This is why you have high TG: too much in blood and all tissues are consuming CARBS" -VLDL starts crashing into other particles FATTY LIVER [predicted by high blood TG] VLDL correlates strongly with TG -if high TG, high VLDL How is VLDL level calculated? Example If TC 6 [LDL 4; HDL 1] How get from TG to 6 TC? Formula: VLDL=TG/2.2 [no evidence for this formula] -there is no other way -When TG too high: Can't use formula VLDL, high carb eaters make a lot of it... -HDL trying to help out...CETP protein also helping HDL is giving up its cholesterol ester & taking in VLDL fat -eventually HDL disappears in its effort to help VLDL So If TG goes UP: HDL DOWN and If TG goes DOWN: HDL UP |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/MXkE8_NdAyQ?si=ce1CsJOGFZGfglIG This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1080 | Dr Ken Sikaris Low Carb Down Under podcast Fats float around in blood in spheres. Inside are -triglycerides [TG] -cholesterol stuck to TG and stuck to fatty acids -outside cover: -phospholipids -apolipoproteins hold it together IN BLOOD many other lipoproteins Lipoproteins: small, dense LDL; HDL; gigantic chylomicrons made after meal to transport fat to liver & other organs... From chylomicrons, tissues take what they need -the remnant goes to liver Liver can make its own fat from sugar -can export fat & cholesterol it made as VLDL cholesterol VLDL cholesterol is full of TG: available for those who burn fat... -as fat removed, it becomes smaller & smaller...becomes LDL -LDL gives up all TG and some cholesterol -should then return to liver, fill up again HDL is MYSTERY -seems to form when lots of cholesterol around to go to liver "THIS IS NATURAL; NO BLOOD VESSELS FOR TG & CHOLESTEROL Liver makes VLDL, gradually transformed; once IDL can return... normal LDL also returns to liver "Those not able to to this are incredibly sick" |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/MXkE8_NdAyQ?si=ce1CsJOGFZGfglIG This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1079 | Dr Nadir Ali Nadir Ali MD podcast Membrane proteins in brain... -amyloid -ApoE -insulin AMYLOID protein involved in neuron growth -attracts white blood cells so brain can protect itself from infection Amyloid Beta is breakdown product of amyloid precursor protein Works well when insulin sensitive In insulin resistance: does not work ApoE4:: designed to eat ancestral diet More at risk of insulin resistance with modern diet Higher LDL Higher heart disease Higher risk Alzheimers Should: intermittent fast, be physical Statins make insulin resistance WORSE ApoE4: Must recover & repair insulin receptors... -restore insulin-glucagon interaction ApoE4 best for this because -no meds can help 1-intermittent fasting 2-severely restrict carbs 3-exercise |||||||||||||||||||||||||||||||| doctorstotrust.com @eatmostlyfat Original Youtube: https://youtu.be/ELxqG-VNvUY?si=1MTKfzvkqGrdGX1L This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1078 | Dr Nadir Ali Nadir Ali MD podcast Body absorbs carbs & protein very differently than fat... -amino acids & sugars go thru portal to liver -fat: into chylomicrons -bypasses liver -thru thoracic duct into general circulation -lipoprotein lipase [insulin-dependent enzyme] -dumps fat into healthy fat cells -or plugs into muscles Once it has dropped off the lipids, it becomes chylomicron remnant -liver designed to deal w/ chylomicron remnant HDL: -primary role is to carry ApoE -donates ApoE to remnant to it can enter liver LIVER role: -clear remnant [cholesterol rich] How well you CLEAR the chylomicron remnant depends on... -ApoE 2, 3, or 4 4? liver sucks it up quickly 3? does okay job 2? does poor job Also happens in BRAIN? -no one has checked! "We know that ApoE 4 are smart" -in higher education -perform tests better "HIGHER LDL ALSO PERFORMED BETTER" |||||||||||||||||||||||||||||||| doctorstotrust.com @eatmostlyfat Original Youtube: https://youtu.be/ELxqG-VNvUY?si=1MTKfzvkqGrdGX1L This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1077 | Dr Nadir Ali Nadir Ali MD podcast Szczepaniak 2009 showed that those with T2D or obese or insulin resistant: stepwise increase in pancreas fat... -liver spews out lots of sugar -insulin UP but -no phase 1 response -glucagon cells covered in fat -so GLUCAGON way UP -CYCLE CONTINUES AS DOCTORS SAY: EAT CARBS! On left: normal response Right: abnormal -no insulin spike -no suppression of glucagon -glucose way UP Cholesterol critical for brain... Brain makes OWN CHOLESTEROL -transported in brain: ApoE -ApoE: protein lipidated by astrocytes -takes cholesterol to different parts of the neuron Types of ApoE: 4, 3, 2 one from each parent... 4: from hunter-gatherer natives -most susceptible to cleavage 3: European 2: Agricultural; Asian; Greek SYNAPTIC CONNECTION BETWEEN BRAIN CELLS NEED CHOLESTEROL -aided by cholesterol-rich areas -receptors sit in cholesterol-rich lipid rafts ApoE is so important in supplying cholesterol, needed for -neuron growth -microtubule stability -clear AMYLOID -synaptic plasticity -SURVIVAL |||||||||||||||||||||||||||||||| doctorstotrust.com @eatmostlyfat Original Youtube: https://youtu.be/ELxqG-VNvUY?si=1MTKfzvkqGrdGX1L This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1076 | Dr Nadir Ali Nadir Ali MD podcast Why is our brain starved for fuel when insulin resistant? -insulin receptors are absent or not working -cause: years of high carb eating: hyperinsulinemia -causing defective receptor HYPERINSULINEMIA stops sugar entry to cell -no protein production -receptor hides in cell -or destroyed by lysosomes With no receptor, brain cannot ignite chemical reactions to -improve synaptic connections between brain cells SO -lose memory -lose brain function Green line: phase 1 response RED LINE: delayed response -even with high insulin IT IS USELESS! LEADS TO HYPERINSULINEMIA Green alpha & red hypertrophied beta cells in Type 2 diabetic... -phase 1 response gone [no control of glucagon] -insulin UP -liver outputs more glucose into bloodstream Sky High Glucagon -muscle breakdown -liver sugar output UP -breaks down PROTEIN! -VLDL output by liver -becomes ectopic fat in pancreas, heart, liver |||||||||||||||||||||||||||||||| doctorstotrust.com @eatmostlyfat Original Youtube: https://youtu.be/ELxqG-VNvUY?si=1MTKfzvkqGrdGX1L This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1075 | Dr Nadir Ali Nadir Ali MD podcast Pancreas: red cells are insulin-producing beta cells... -green produce glucagon Type 1 diabetics have no beta cells Type 2 diabetics have hypertrophied beta cells [ineffective] Both alpha & beta cells close to each other... Dr Unger asked: is this for a purpose? Paracrine signaling: means local control [endocrine means signal comes from pancreas] -insulin created in pancreas to have effect on brain Insulin begins in beta cells -highest concentration at alpha cells making glucagon "Role of insulin is to SHUT DOWN GLUCAGON" [from Dr Roger Unger] From Dr Roger Unger: When insulin reaches liver, it is diluted 10x... -when enters circulation: diluted 100x Injected insulin can NEVER DUPLICATE THIS [liver will never see this normal concentration of insulin] From Dr Roger Unger: As there are slight fluctuations in sugar -beta cells release insulin in phase 1 response -in a squirt to suppress glucagon Continues to suppress glucagon thru phase 2 When not eating, glucagon levels are high... Tells liver: put out sugar -burn muscles, convert to sugar -take glycerol from fat & convert to sugar -take fatty acids & convert to KETONES Paracrine action between alpha and beta cells when eating... -insulin tells : -use & store sugar -build new protein -store fat When NOT EATING glucagon tells: -release sugar from liver -break down protein to make sugar -release fat for sugar & ketones |||||||||||||||||||||||||||||||| doctorstotrust.com @eatmostlyfat Original Youtube: https://youtu.be/ELxqG-VNvUY?si=1MTKfzvkqGrdGX1L This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1074 | Dr Nadir Ali Nadir Ali MD podcast Dr Nadir Ali: sugar map of the brain... Normal: good uptake Alzheimers: poor Adding sugar Normal: 150mg/dL -improved memory Alzheimers: 225 Brain requires insulin to drive energy in Insulin receptor as insulin binds to it... -cell activates GLUT4 channel to bring sugar in -burn for energy -or, convert to glycogen Same cell will also -insulin receptor substrate 1 [IRS-1 converts sugar into mTOR] -making protein Brain cell has MANY insulin receptors in hippocampus... -receptor brings in fuel AND creates chemical reactions -new proteins formed that FACILITATE MEMORY: Synaptic Plasticity When you eat high carb, insulin tells liver -get ready to store sugar or use it Tells muscle -amino acids are coming -build new protein Tells fat cells -store fat in fat cells Tells brain "I love this sugar! Let's do it again!" |||||||||||||||||||||||||||||||| doctorstotrust.com @eatmostlyfat Original Youtube: https://youtu.be/ELxqG-VNvUY?si=1MTKfzvkqGrdGX1L This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1073 | Dr Paul Saladino w/ Dr Alo Paul Saladino MD podcast Study in humans -decreasing saturated fat -INCREASING linoleic acid [seed oil] Results increased oxidized LDL & Lp(a) [no cardiologist would say this is good] These are signs of inflammation -bad, bad cardiovascular outcomes with the seed oils Is Dr Saladino keeping you AWAKE doctor?? Study in humans... Linoleic acid & colostrum & child cognition at 2 & 3 years Results too high levels of linoleic acid in colostrum: associated w/ POOR CHILD DEVELOPMENT at 2 & 3 Diet & sex hormones in healthy men... -increase in seed oils: decrease in sex hormones NIH Study -lowering linoleic acid reduces oxidized LA metabolites in humans Clear: consuming more seed oils causes accumulation of linoleic acid in our adipose tissue AND in our LDL particles LDL enriched w/ LA is more likely to be oxidized in sub-endothelial space Wrapping up debate part 1: -with LDL enriched by LA -like a bomb -LDL much more likely to become oxidized in the macrophage environment...into sdLDL Brown & Goldstein showed: macrophages do not take up native LDL in sub-endothelial space -ONLY TAKE UP OXIDIZED LDL "Oxidized LDL is the real problem, NOT NATIVE LDL" Dr Alo: "I am not a vegan or vegetarian. Love my ribeyes. I don't eat steak & butter every day... My LDL is 46" Dr Saladino: Do you take med?? Dr Alo: "yes, Zetia & Crestor [statins] It is not a problem. My people [Youtube followers] all know this" |||||||||||||||||||||||||||||||| doctorstotrust.com paulsaladinomd.co Original Youtube: https://youtu.be/kAvgNR7O79c?si=EUtJzEBgzPuNMtJm This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1072 | Dr Paul Saladino Paul Saladino MD podcast Dr Paul Saladino with Dr Alo... "You challenged me to show a study of how linoleic acid was harmful to health" Literature shows linoleic acid: -increases LDL oxidation -oxidized LDL in sub-endothelial is a very bad thing Jerusalem Nutrition Study -more seed oils=more oxidation -more LA break down products -LESS LA=LESS BREAK DOWN "We know, when more linoleic acid in LDL particle, that particle is more susceptible to oxidation Study Increased saturated fat will increase LDL will decrease Lp(a) [LDL+Lipoprotein=Lp(a) very high risk for heart disease] Decreased sat fat & increased LA "WILL INCREASE Lp(a) & oxidized LDL" -increased LA decreases testosterone Study in humans... soybean consumption INCREASES Lp-PLA2 -involved in cleaving oxidizing phospholipids & LDL Study strong correlation between linoleic consumption & MACULAR DEGENERATION and mothers breastfeeding: LA correlates negatively with kids' cognitive & motor development "Consistently, researchers are looking at serum levels of LA, when the only seriously good metric to judge LA in diet is: adipose tissue! to Dr Alo: "You posted study on Twitter...the studies in that pooled analysis: Only 3 looked at LA in adipose tissue--and in none of those 3 was LA associated with improved cardiovascular outcomes. "There are studies: LA in adipose tissue: worse outcomes” |||||||||||||||||||||||||||||||| doctorstotrust.com paulsaladinomd.co Original Youtube: https://youtu.be/kAvgNR7O79c?si=EUtJzEBgzPuNMtJm This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1071 | Dr Ken Sikaris Low Carb Down Under podcast PCSK9 protein Summary so far: Cholesterol is good LDL is good Modified LDL is BAD -sdLDL TG greater than 1.5mmol/L [133 mg/dL] -glycation [HbA1c greater than 5.6%] -oxidation Lipoprotein(a): Lp(a) = LDL + Apolipoprotein(a) with tail -tends to stay in blood longer -higher Lp(a)=higher risk of cardiovascular event -no meds to change it -unsure what low carb high fat does -in 1/3: raises cholesterol -first thing Dr Ken checks to see of sdLDL or other 3rd protein in LDL receptor: PCSK9... -normally, LDL combines to receptor on adipocyte -taken in by cell; destroyed -receptor captures more LDL [want it all back after 1 week] PCSK9 tags LDL & receptor "after destroying LDL, destroy receptor also" -so less LDL comes back out of blood stream Higher PCSK9 means higher cholesterol in blood Levels of PCSK9 in blood ranges from 100 to 500... -if taking off receptors, so LDL is hanging around in blood -will become small & dense About 5% of LCHF patients [low carb high fat] have exaggerated response to diet -Key? Is their cholesterol small & dense? -we don't know % of PCSK9 in population Worry about cholesterol? NO -only sdLDL or modified LDL -reduce by LCHF -need to learn more about: -Lp(a) -PCSK9 |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/OyzPEii-wo0?si=U26oRJtdxh840ToX This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1070 | Dr Ken Sikaris Low Carb Down Under podcast Blood lipid tests show LDL... [in green here on the left] -red: small, dense: don't want! -higher red = higher risk of atherogenic type of cholesterol Generally: if you have -high cholesterol, you have -high LDL "Don't care about LDL, just about small, dense LDL" -THAT RELATIONSHIP IS VERY POOR Could have cholesterol of 8 with virtually NO small, dense LDL... -or, 8 with 30% small, dense Get tested...or: -predict from TG level -if TG below 1 mmol/L [88.5 mg/dL] have no sdLDL -if over TG 1.5 THEN HAVE sdLDL: TROUBLE -study 2001: TG aboce 1.2 sdLDL begin to RISE Better predictors than TG levels... -LDL to HDL or -Total cholesterol to HDL both predict better than TG Study, 2008 -TG to HDL best predictor of obstructed blood vessels Study, 2014 TG -highly variable from test to test -not evenly distributed HDL more stable [HDL is better marker of TG than TG alone!] Effective treatments to reduce TG will decrease sdLDL Study, 2008 -most severe restriction of carbs = -most reduction of TG -most increase of HDL Study, 2015 -lower carb = lower sdLDL |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/OyzPEii-wo0?si=U26oRJtdxh840ToX This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1069 | Dr Ken Sikaris Low Carb Down Under podcast 1 in 10,000 have abnormality that prevents liver from taking up the LDL... Familial Hypercholesterolemia -LDL is accumulating in tissues: -eyes, tendons, blood vessels What type LDL? -small, dense LDL "It is a disease, not of high cholesterol, but small, dense LDL" For those with Familial Hypercholesterolemia... -statins improve their health -reduce small, dense LDL Small, dense LDL can get into blood vessel walls... -forms plaque -changed by glycation & oxidation -taken up by macrophages -become engorged That is the PLAQUE that causes heart disease Summary -VLDL becomes LDL -should return to liver -if not, becomes small, dense -into vessel walls -becomes PLAQUE Paper 2015: "When oxidize LDL, macrophages go crazy!" Paper 2010: Glycating LDL [sticking sugar onto]... "We know sticking sugar onto hemoglobin relates to heart disease" Paper 2008: sticking sugar onto macrophages makes them go crazy-- accumulates in vessel walls Normally LDL is large, good... -high TG makes small, dense -into vessel wall -glycated & oxidized -TROUBLE |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/OyzPEii-wo0?si=U26oRJtdxh840ToX This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1068 | Dr Ken Sikaris Low Carb Down Under podcast LDL particle at liver: controlled by -ApoB [protein] -LDL receptor -PCSK9 regulator Paper, 1972, looked at abnormalities in receptor... Normally, LDL disappears in week or two from blood If receptor abnormal, it hangs around in blood 2x [just like in diabetes] Too much fructose, end up with too much TG in liver... -exported as VLDL -can interact with HDL [using CETP] which wears out HDL] -patient w/ high TG & VLDL will wear out HDL "High TG ALWAYS results in lower HDL" & high TG interacts w/ LDL -result: small, dense LDL due to high TG so: long term LDL in blood Normal big, buoyant LDL [study]: if stay big, risk of heart disease: NO CHANGE if increasing volume of small, dense LDL: heart disease risk UP 4x "Not LDL; but small, dense LDL causing heart disease" |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/OyzPEii-wo0?si=U26oRJtdxh840ToX This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1067 | Dr Ken Sikaris Low Carb Down Under podcast Cholesterol: Greek word meaning bile / solid... -without cholesterol we would not be able to absorb fat using bile -without cholesterol, cells would fall apart -precursor for vitamin D -without it, sunlight pointless -all steroid hormones made from it -liver makes 85% of it [idea that cholesterol in diet as bad has died] Important lipoprotein particles of cholesterol in blood... -liver makes VLDL -full triglycerides & fat -in blood, TG taken out to muscle and other -smaller & smaller until cholesterol-rich LDL particle [VLDL: TG rich] [LDL: cholesterol rich] -NORMALLY now LDL goes straight back to liver Russian scientist fed cholesterol to rabbits, caused plaques... "Not in rats. Don't know if valid for humans" Ansel Keys, who put the cloud over cholesterol... -said the problem was fat Keys' graphs: -higher cholesterol; higher heart disease That study was corrupt -recent studies found opposite Higher saturated fat=less likelihood of dying Important lipoprotein particles of cholesterol in blood... -liver makes VLDL -full triglycerides & fat -in blood, TG taken out to muscle and other -smaller & smaller until cholesterol-rich LDL particle [VLDL: TG rich] [LDL: cholesterol rich] -NORMALLY now LDL goes straight back to liver |||||||||||||||||||||||||||||||| doctorstotrust.com lowcarbdownunder.com.au Original Youtube: https://youtu.be/OyzPEii-wo0?si=U26oRJtdxh840ToX This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1066 | Dr Anthony Chaffee w/Coach Dant How To Carnivore podcast Coach Dant: I owned a cross-fit gym and did 3 workouts a week... -was paleo whole 30: no processed foods; no alcohol -just meat & plants -had many pulled muscles, torn joints, etc. Went full 100% carnivore At 3 months: performance returned -after dipping down initially Strength numbers started climbing Finally, aches & pains went away Increased volume & recovery was better The other reason, besides pain, for me to go carnivore was... -tired of having to get 'fat to get strong' and -get 'weak to get lean' Whenever I wanted to cut, I had to tell self: all my performance numbers will fall Then, work on getting strong again, have to bulk up & look like a slob with this got ripped and stronger together Question: What was your first approach with clients? -freak out! 'no veggies?' -started with helping them understand: -okay to eat more animal foods -don't have to fear fat Ran 10 week challenges: carnivore beginning -for my experience working with this -worked for all of them -no calorie restriction -just: add more meat & continue workouts Had tremendous results |||||||||||||||||||||||||||||||| doctorstotrust.com howtocarnivore.com Original Youtube: https://youtu.be/E79M0fZ4YHo This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1065 | Dr Anthony Chaffee w/Coach Gant How To Carnivore podcast Dr James Salisbury, Scott, NY born 1823 Elma Stuart wrote book describing Dr Salisbury's diet... -1880's "What Must I Do to Get Well? -clinical report of what he had been doing for 10+ years to heal people -for most conditions, solution was: 'eat stewed beef' -don't eat anything else [Elma was bedridden with Fibromyalgia; took 1 year for full recovery on this diet] Dr Chaffee: "I had thought the name of the Salisbury Steak came from the town in England" -beef was ground in way to filter out all gristle -body absorbed 100% of nutrients -for Crohn's and ulcerative cholitis, which had NO MEDS back then -without meds, people were in horrible trouble with digestive diseases -with no gristle, could absorb everything in small intestine with no waste -letting bowels rest & heal We do same today: if have bowel disease, bowel surgery, put on low-residue diet, so no waste -no fiber whatsoever "Idea that fiber is good for you is contradicted by that" "When bowel problems: NO FIBER!" "But when healed, 'better eat fiber!" 1980s: told: eat fiber for 2 reasons -can't digest it; no nutrition, bulky so it will stress your receptors; make brain think you are full "You body is not that dumb!" Second: -doe to low fat eating, lots of constipation -"You should eat more fiber" -whole idea was: can't digest it, can't get energy or nutrition from it SO WHY EAT IT? |||||||||||||||||||||||||||||||| doctorstotrust.com howtocarnivore.com Original Youtube: https://youtu.be/E79M0fZ4YHo This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1064 | Dr Ken Berry Ken Berry MD podcast You can reverse your type two diabetes: on your own, for free. If you are currently on medication, first, check with your doctor. We could call this chronic carbohydrate overdose. It is not your fault, but it is your problem. Step one: stop all sugar consumption: that you added, or food company added. These processed items are not food they spike blood sugar. Step two: stop eating all grains, like wheat, corn, oats, rice. Any grain converse immediately in the sugar, even in your mouth Step three: remove all vegetable oils, processed, seed oils, from diet, including: canola, soy bean, sunflower, safflower, peanut, corn. All are inflammatory. All have high omega6 to 3 ratio, all oxidize rapidly. Step four: eat lots of fatty meat. Fat and protein do not raise blood sugar or insulin very much. They contain tons of vitamins and minerals. Step five: Get your carbs if you want some from above ground, non-starchy vegetables. Carbohydrates: how much to eat and still reverse type two diabetes. Some people can eat a total each day of 100 g. Others 50 g, others 20 g. |||||||||||||||||||||||||||||||| doctorstotrust.com drberry.com Original Youtube: https://youtu.be/uR2ksqDy5os?si=QcM5MOtIVIPXGnT- This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1063 | Dr Michael Shellenberger Univ of Texas at Austin podcast Michael Shellenberger at Univ of Texas Austin... -climate change is destroying future -planet has warmed 1.1 C -killing Great Barrier Reef -pandemic police killing black people -murders of trans people up 2x -carbon peaked in GB, US, France 50 years ago -past decade total emissions declined -US natural disaster deaths declined to a few hundred -worldwide dropped over 90% -costs of natural disasters has declined -total global weather & climate disasters have DECLINED -Great Barrier Reef: highest coral in 36 years [since tracking began] "Why do people who say they care... allow schizophrenics smoke fentanyl and their legs rot on the streets?" -amputated -put back on street We spend 10x more than before for these outcomes -why do we call this compassionate? No racial differences in police killings -armed, not armed, justified... -everyone knows data is right Roland Fryer, Harvard University Police killings of African-Americans: -1950s 217/year -today 157/year -FBI data for top 58 US cities Trans people homicides -disproportionally or on the rise? -data is not known "Yet CBS News irresponsibly says otherwise...sets bad baseline" Why do we get this so tragically, unethically wrong? |||||||||||||||||||||||||||||||| doctorstotrust.com @GrassrootsArmy Original Youtube: https://youtu.be/E79M0fZ4YHo This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1062 | Dr Georgia Ede Levels podcast Dr Dom D’Agnostino: French study, 31 treatment-resistant, seriously mentally ill patients on keto diet... -bi-polar; major depression; schizophrenia -75% animal fat; 20% animal protein; 5% carb -100% improvement in symptoms -43% achieve clinical REMISSION -96% of patients lost weight -64% discharged on less meds This was observational study, no control group -but actual results from clinic in Toulouse, France Patients had severe, chronic mental illness -at clinic for decades -had NOT RESPONDED TO ALL TYPES OF interventions: -meds -psychotherapies -support & repeated hospitalizations -some: electric convulsive therapy "He had seen young family member with epilepsy & autism respond well to keto in just a few weeks" Decided to try this brain-healthy approach Patients stayed in hospital, some for long period of time... -this had NEVER BEEN DONE BEFORE -28 of 31 stayed on diet -all improved "43 achieved clinical REMISSION; you do not see that in typical psychiatric care" -did not put half the patients into control -clinician trying to help his long term patients "We published as retrospective study since these results were so outstanding" It has inspired more rigorous research |||||||||||||||||||||||||||||||| doctorstotrust.com levelshealth.com diagnosisdiet.com Original Youtube: https://youtu.be/kBjBEjV0vqU?si=xN1CLWKC6vD3sQ-8. This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.

http://www.DoctorsToTrust.com presents episode 1061 | Dr Georgia Ede Levels podcast CGM [continuous glucose monitor]... -very helpful for my patients Example: bowl of oatmeal for breakfast -can have profound impact on blood sugar "We are told all the time: 'oatmeal is brain healthy food'" NOTHING COULD BE FURTHER FROM TRUTH In my new book [Change Your Diet, Change Your Mind] Example of CGM Tracing -eating all types of oatmeal -had to remove all oatmeals from diet to stabilize her mood She was able to take control over brain health -rather than wondering why having panic attack -or, taking medication "Why not start with food FIRST?" 9 times out of 10, will work! Can see connection between what you eat and how you feel Fasting insulin is one of best lab tests for checking mental state... -ideally it is 6 or below [US measure] "When at Smith College, would routinely see young women with levels in 20s and 30s" -when they dropped their carbs, predictably their insulin would drop in a few weeks. -same with triglycerides Almost always drop like a stone on low carb -even by hundreds of points in few weeks -not everyone, but vast majority |||||||||||||||||||||||||||||||| doctorstotrust.com levelshealth.com diagnosisdiet.com Original Youtube: https://youtu.be/kBjBEjV0vqU?si=xN1CLWKC6vD3sQ-8. This site will never use corruptible, epidemiological survey research as causal science. For each short/sharable video, the original Youtube links are provided None of this content is intended to be individual, personalized medical advice. We hope you find value for yourself in these short videos & find them easy to share with loved ones! DISCLAIMER The DoctorsToTrust videos are for general informational purposes only and do not constitute the practice of medicine, nursing or other professional health care services, including the giving of medical advice, and no doctor/patient relationship is formed. The use of information on this podcast or materials linked from this podcast is at the user’s own risk. The content of this podcast is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Users should not disregard or delay in obtaining medical advice for any medical condition they may have and should seek the assistance of their health care professionals for any such conditions.