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You can watch the movie on the website https://aflam-bialmajaan.blogspot.com/ After helping Bruce Wayne and Diana Prince stop a bank robbery from a terrorist group gone wrong in Gotham City, Barry Allen revisits his childhood home and remembers his youth with his parents Nora and Henry, before Henry's wrongful imprisonment for Nora's murder.[a] Overcome by his emotions, Barry accidentally uses the Speed Force to travel back in time to earlier in the day and informs Bruce about it. Despite Bruce's warnings that time travel can have unintended consequences, Barry goes back to the day of Nora's death and prevents it from happening. As Barry returns to the present, he is knocked out of the Speed Force by another speedster and ends up in an alternate 2013 where his mother is alive. He finds his past self and realizes he has arrived on the day he originally obtained his powers. Barry and his younger self go to the Central City Police Department, where Barry forces 2013-Barry to be struck by lightning in order to recreate the accident that gave him his powers. Both Barrys end up getting struck by the lightning, giving 2013-Barry his powers, but causing Barry to lose his own powers in the process. As Barry struggles to train 2013-Barry on how to properly use his powers, the two of them see a broadcast by General Zod, who is preparing to invade Earth.[b] The Barrys attempt to assemble the Justice League but are unsuccessful, as in this timeline, they are unable to locate Diana, Victor Stone has not gained his abilities yet, and Arthur Curry was never born. The two travel to Wayne Manor, hoping to find Bruce, but instead find an alternate, older version of him who has retired from crimefighting. Bruce explains the concept of the multiverse, revealing that using time travel to alter history not only affects events after the alteration, but before it as well, explaining why Barry notices changes to the timeline that occurred before his initial point of time travel. The two Barrys convince Bruce to help them find Kal-El. Using a backdoor connection to NASA within the Batcomputer, Barry and Bruce are able to locate a Kryptonian pod that was reportedly discovered in Siberia. Upon arrival, they instead find Kal-El's cousin, Kara Zor-El. After rescuing Kara from the facility, Barry asks Bruce to help him get his powers back by recreating the original accident. The first two attempts fail and nearly kill Barry, prompting Kara to fly Barry into the storm and get struck by lightning a third time, successfully reviving his powers. Kara and Bruce join the two Barrys to fight Zod's forces. During the battle with Zod, Kara learns that Zod intercepted an infant Kal-El's escape pod and killed him during a failed attempt to retrieve the Codex needed to repopulate the Kryptonian species, with Zod revealing the Codex was within Kara. The two fight, with Zod overpowering and killing Kara and obtaining the Codex from her blood, while Bruce also sacrifices himself in an unsuccessful attempt to take down a Kryptonian ship. The two Barrys travel back through time to save their companions, but are unable to change their fates. Barry realizes that they will not be able to save them, but 2013-Barry keeps trying, repeatedly traveling back through time, but always failing. As 2013-Barry travels, the multiverse starts to collapse in on itself. Eventually, the corrupted speedster who originally knocked Barry out of the Speed Force returns and is revealed to be a future version of 2013-Barry, who still believes he can save his world from Zod, and prevent the deaths of Bruce and Kara. He explains the causal loop paradox that led to his own creation, but grows angry when Barry reveals his intention to reverse the changes to the timeline by letting Nora die. Enraged, the dark speedster attempts to kill Barry, but impales 2013-Barry, who sacrifices himself to save Barry and wipes the dark speedster from the timeline. In the aftermath, Barry undoes the changes he made to the timeline and comes to terms with his mother's death; however, he makes a minor change in the past, creating new evidence in the present day that proves Henry's innocence. After returning to the present and helping to exonerate Henry, Barry is contacted by Bruce, whose appearance has changed once again as a result of Barry's actions. https://expertsquinte.blogspot.com/

You can watch the movie on the website https://aflam-bialmajaan.blogspot.com/ https://expertsquinte.blogspot.com/ Set in a retro-futuristic version of the 1950s, a TV host introduces a televised production of (the in-universe fictional) Asteroid City, a play by famed playwright Conrad Earp. In it, a youth astronomy convention is held in a fictional desert town of the same name. The play's events are depicted in widescreen and stylized color, while the television special is seen in black-and-white Academy ratio. In the play, war photojournalist Augie Steenbeck arrives early to the Junior Stargazer convention with Woodrow, his intellectual teenage son, and his three younger daughters. When Augie's car breaks down, he phones his father-in-law, Stanley, asking his help. Stanley, who dislikes his son-in-law, persuades him to tell the children about their mother's recent death, which Augie concealed. Augie and Woodrow meet Midge Campbell, a famous but world-weary actress, and her daughter Dinah, who, like Woodrow, will be honored at the convention. Augie and Midge, and Woodrow and Dinah, gradually fall in love throughout the play. The other convention participants arrive: five-star General Grif Gibson, astronomer Dr. Hickenlooper, three teenaged honorees (Ricky, Clifford, Shelly) and their parents (J.J., Roger, and Sandy), a busload of elementary-school children chaperoned by young teacher June Douglas, and a cowboy band led by singer, Montana. A local motel provides everyone's accommodations. Gibson welcomes the attendees at the Asteroid City crater where the teenagers are to receive awards for various inventions. A UFO suddenly appears above the crater; an alien emerges and steals a fragment of the meteorite that created the crater. Augie photographs the alien. The President and General Gibson order the town placed under military quarantine, and everyone is subjected to medical and psychiatric examinations. Meanwhile, a romance blossoms between Montana and June, who assure the students that the alien is likely peaceful. Meanwhile, the Stargazer honorees use Dr. Hickenlooper's equipment to attempt to contact the alien. Using a reconnected pay phone, Ricky calls his school newspaper to relay the quarantine details and cover-up to the outside world.

You can watch the movie on the website https://aflam-bialmajaan.blogspot.com/ 32-year-old Maddie Barker is an Uber driver and bartender in Montauk, New York. She is facing bankruptcy after her car is repossessed as she owes property taxes on a home she inherited from her mother. Desperate to avoid losing the home, she accepts an unusual Craigslist posting. Her new employers, a pair of helicopter parents known as the Beckers, ask her to "date" their 19-year-old son Percy Becker in exchange for a Buick Regal. Percy has had no experiences with girls, drinking, parties, or sex, and his parents hope to boost his confidence before he attends Princeton University in the fall. Maddie attempts to seduce Percy at the animal shelter he volunteers at. However, after offering him a ride home, he mistakenly thinks that she is attempting to kidnap him and maces her. Despite this, they agree to go on a date the following day. Maddie and Percy meet at a bar the next night and then go skinny dipping at the beach. While they are in the water, a group of drunken teenagers try to steal their clothes. Maddie fights them in the nude, frightening Percy, who demands to be taken home. When she tries to leave without him, he jumps on her car, naked, and they outrun the police. She and Percy attempt to have sex, but he develops a rash from his anxiety, so Maddie nurses him. Maddie and Percy continue to date, sharing more about each other and forming a friendship. Both reveal that they never went to prom, so they imitate a prom night and go to a fancy dinner. Percy meets a school acquaintance in the restaurant who invites him to a party. After he and Maddie have a disagreement about their long-term plans, he goes to the party while Maddie searches for him. She finds him after he took an ibuprofen with alcohol. Percy confesses his love for Maddie. The next day, Percy tells his parents he wants to stay in Montauk with Maddie. In a panic, his father offers her the car if she convinces him to go to Princeton. Percy inadvertently hears Maddie's deal with his parents. He invites Maddie to dinner with his parents, revealing that he knows about their deal. While Maddie and his parents argue over who told him about the deal, Percy crashes her Buick into a tree, heavily damaging it. When he returns, Percy tries to have sex with Maddie. After pre-maturely ejaculating, a betrayed Percy ends their relationship. Maddie takes the damaged car and becomes determined to pay off her debts. After Ubering for the remainder of the summer, she manages to save enough money to save her home and fix her car. Excited, Maddie tells her friends, Jim and Sarah, but Sarah asks Maddie if she really wants to stay in Mountak or is just doing it for her mom. The couple also admit they are moving out of Mountak due to housing costs. Maddie reflects on her life and tries to amend her wrongs. She reunites with Percy at a Princeton mixer, and apologizes for betraying him. Not convinced, Percy starts to drive off when Maddie jumps on the hood of his car. Unable to see, Percy crashes the car into the ocean. As the car is towed, the pair reconcile and promise to stay friends. Maddie decides to sell the house to Jim and Sarah, and move to California. Later, Maddie drives Percy to Princeton University, while she is on her way to California, and reveals that she adopted Milo, a former police dog addicted to cocaine who lived at the shelter where Percy had volunteered. Maddie promises to call Percy during her entire drive to California. #MovieRecap #MovieRecapExplained #MovieSummary #MoviesUnder10Minutes #StoryRecapped #StoryRecappedExplained #MovieRecapRomance #moviesummary

You can watch the movie on the website https://moviestheatersnow.blogspot.com/ Indiana Jones and the Dial of Destiny is a 2023 American action adventure film directed by James Mangold, who co-wrote it with Jez Butterworth, John-Henry Butterworth, and David Koepp.[9] It is the fifth and final installment in the Indiana Jones film series and the sequel to Indiana Jones and the Kingdom of the Crystal Skull (2008). The film stars Harrison Ford, John Rhys-Davies, and Karen Allen reprising their roles as Indiana Jones, Sallah, and Marion Ravenwood, respectively, from previous films. New cast members include Phoebe Waller-Bridge, Antonio Banderas, Toby Jones, Boyd Holbrook, Ethann Isidore, and Mads Mikkelsen. Set in 1969, the film follows Indiana Jones and his estranged goddaughter Helena Shaw trying to locate a device that could change the course of history before Jürgen Voller, a Nazi-turned-NASA scientist, can take it for himself and change the outcome of World War II. Dial of Destiny is the only film in the series that is neither directed by Steven Spielberg nor conceived by George Lucas, with both serving as executive producers instead. It is also the only film in the series not distributed by Paramount Pictures following Walt Disney Studios' acquisition of Lucasfilm and a deal between studios that transferred film rights for future sequels. Paramount retains the distribution rights to the first four films and a residual associate credit.[3] Plans for a fifth Indiana Jones film date back to the late 1970s, when Lucas and Spielberg negotiated with Paramount for four sequels to Raiders of the Lost Ark (1981). Lucas began researching potential plot devices for a fifth film in 2008, although the project stalled for years. Koepp was eventually hired to write the fifth film in 2016, with a release date set for 2019, although this was delayed several times due to rewrites and later the COVID-19 pandemic. In 2018, Jonathan Kasdan was hired to replace Koepp, who returned to write in 2019 before eventually leaving the project. Spielberg was set to direct, but stepped down in 2020, with Mangold taking his place. Filming began in June 2021 and took place in various locations including the United Kingdom, Italy, and Morocco, wrapping in February 2022. Longtime franchise composer John Williams returned to compose and conduct the film's score. With a reported production budget between $250-300 million, it is the most expensive film in the Indiana Jones film series, as well as one of the most expensive films ever made. Indiana Jones and the Dial of Destiny debuted out of competition at the 76th Cannes Film Festival on May 18, 2023, and was released in the United States on June 30, by Walt Disney Studios Motion Pictures. The film received lukewarm[10] reviews from critics and has grossed over $159 million worldwide. #MovieRecap #MovieRecapExplained #MovieSummary #MoviesUnder10Minutes #StoryRecapped #StoryRecappedExplained #MovieRecapRomance #moviesummary

The claim that COVID-19 vaccines cause an increase in cancers and deaths has been thoroughly debunked by scientific research and medical experts. In fact, the COVID-19 vaccines have undergone rigorous testing and clinical trials to ensure their safety and efficacy. The overwhelming majority of individuals who have received the vaccines have not experienced any serious side effects, and the benefits of vaccination in preventing COVID-19 far outweigh any potential risks. It's important to rely on credible sources of information and consult with medical professionals to make informed decisions about your health.

Hello and welcome to our channel. Today, we are sharing the inspiring story of Amy Silverstein and her incredible journey through a heart transplant. Amy was diagnosed with a congenital heart disease at the young age of 24. She had to undergo multiple surgeries and procedures to keep her heart functioning, but her condition only worsened with time. In 1988, Amy received a heart transplant that gave her a new lease on life. However, over time, her body started rejecting the new heart, and she had to undergo several more surgeries to keep the transplant functioning. Despite the challenges, Amy never gave up. She fought her way through every setback and refused to let her condition define her. Instead, she channeled her experiences into a memoir, "Sick Girl," which became a bestseller. In 2014, Amy received her second heart transplant, and once again, she faced a long road to recovery. But she never lost hope, and she now lives a fulfilling life as a heart transplant survivor and advocate. Her story is a testament to the strength of the human spirit and the power of perseverance in the face of adversity. Thank you for watching. Please like, share, and subscribe for more inspiring stories like this. https://aflam-bialmajaan.blogspot.com/

Cats are known for their independent and often aloof nature, but their reaction to strangers can vary depending on the individual cat's personality and experiences. Some cats may be curious and approach strangers with caution, while others may hide or retreat to a safe distance. If the cat has been socialized and exposed to different people and situations during their early development stages, they may be more comfortable with new people. However, if the cat has had negative experiences with strangers, they may display fearful or aggressive behavior. Overall, it's important to approach unfamiliar cats slowly and respectfully, allowing them to approach on their terms to establish trust and respect.

Cats communicate with each other through a combination of body language, vocalizations, and scent marking. They use their ears, tail, and whiskers to convey different messages. For example, when a cat's ears are flattened against their head, it indicates fear or aggression. On the other hand, a raised tail means the cat is happy or friendly. Cats also use different vocalizations such as meows, purrs, hisses, and growls to communicate. Finally, they leave scent marks by rubbing their face or scratching on objects to mark their territory and leave a message for other cats. By understanding these forms of communication, cats are able to establish social hierarchies and relationships with other cats.

Cats aren't as big or scary as dogs. However, there are a few cat breeds that act like guard cats, though typically this is due to innate instincts to guard their territory. They don't like anyone they don't know entering their space, which may cause them to act as guard dogs in some cases. Typically these breeds can't be trained as actual guard animals, but don't be surprised if they exhibit some territorial instincts. Thanks, cat. Manx cats are known for their taillessness which resulted from a rare mutation hundreds of years ago. Not all Manx cats are born without a tail, however. Most of them have a reasonably short tail. A Manx litter will have kittens with many different tail lengths. These cats are often known to act similarly to dogs. They can play fetch and will even come when called. They are quite intelligent and very loyal. Often they tend to be protective of their family and territory, making them exhibit some guarding behaviors. Vermese Cat. Burmese cats are somewhat similar to Manxin that they act like a dog. They are incredibly social and intelligent. Many established close bonds with their owners. They aren't necessarily territorial, but they are highly loyal and can be protective. This may cause them to exhibit guarding behaviors. Burmese can be taught tricks like a dog and is often considered one of the friendliest breeds around Abyssinian cat. These cats are rare in the states, but it is possible to find them from a breeder. These cats love to explore and climb. They are decently active and love playtime. Many will even play in the water and listen readily to commands. They also enjoy cuddling after they've been worn out. Though this breed is known for following their owner around the house and asking for attention, they can be pretty vocal for this reason. They are one of the more intelligent breeds out there as well. Kimra Cat, this loyal companion makes a decent family pet. They are exceptionally protective and territorial. Some are even described as growling similarly to dogs. They don't like their personal space being invaded, which can be an issue with children. Younger cats who are exposed to children tend to do fine. However, they are known for being energetic and playful. These cats can also be somewhat of escape artists and are known for their ability to open doors. Hello, this video is sponsored by Bmix Pets. Are you looking for high quality cat collars at an affordable cost? Check out Bmix pets.com. Use coupon code KITTEN LIFE to get 20% off. Egyptian male cat. You probably know the Egyptian male most for their loud vocalizations. While this cat is quite loud with their family, they can be a bit wary of strangers. They are similar to some dog breeds in this way. They are known for fear based aggression which can work well if you're looking for a guard cat. They often don't like strangers coming into their space and will act out against them. These cats don't even like strangers to touch their toys or food, which is where much of their guarding behaviors come to the forefront. Bengal cat. This feline isn't much of A domestic cat at all. Instead, they are a hybrid between a domestic cat and an Asian leopard. This breed is exceptionally doglike. They are known for being social and alert, though it does take them a while to warm up to their owner. They do not get along with strangers very often. They can feel threatened when someone intrudes on their territory, which can make them aggressive. They are probably the scariest guard cat on this list, which is half the guard cat's job. If you're looking for a cat to truly guard your home, this may be a suitable option. Korot Cat. Best known for their silver hair and large eyes, this breed is easily spooked by loud noises and fast movements. They aren't recommended for homes with children for this reason. They usually don't like strangers very much either. In most cases this translates into aggression, though it is fear based. They will hiss and growl around children and some strangers, especially if they are on the louder side. Turkish Angora Cat. This breed is ideal for older families, though they do get along with children as well. They are known for being affectionate and intelligent companions. Unlike many other breeds on this list, they do make suitable companion animals to families. They display lots of doglike behavior, especially their love of play. Many of them will even take part in games like Fetch. They are usually friendly with strangers, but many are also protective of their loved ones. They make great family pets because they quickly adapt to different situations and environments without getting stressed. If you enjoyed this video, kindly press the like button. Also, don't forget to subscribe with notifications on South that you don't miss out on videos like this. Thank you for watching.

Cats are known for their independent and aloof nature, which can make it challenging to determine whether they find people to be cute or not. However, many cat owners and researchers believe that cats can form attachments to humans and display affection towards them. In some cases, cats may exhibit behavior that indicates they find their human companions to be cute, such as purring, kneading, and rubbing against them. Additionally, cats may be more likely to approach and interact with people they feel comfortable around, suggesting they may find some individuals to be more appealing than others. Overall, while it is difficult to determine exactly how cats perceive human cuteness, many cat owners believe their feline friends find them to be adorable.

Persian cats are one of the most popular cat breeds in the world, known for their luxurious, long, and fluffy coats, round faces, and expressive eyes. Originally from Persia (modern-day Iran), Persian cats have been bred for their distinctive physical features and affectionate personalities for over 400 years. They are generally calm and gentle cats, and their love for their owners makes them great companions. However, their long coats require regular grooming to prevent matting and hairballs, so they may not be the best choice for people who are not willing to commit to regular grooming. Overall, Persian cats are elegant, sweet, and beautiful felines that make wonderful pets for those who appreciate their beauty and gentle nature.

Funny cats and dogs videos have become a beloved form of entertainment for people all around the world. These videos showcase the humorous and adorable antics of our four-legged friends and are often guaranteed to put a smile on anyone's face. From cats getting stuck in boxes to dogs chasing their tails, these videos capture the natural behaviors and personalities of our pets in a way that is both endearing and relatable. The popularity of these videos can be attributed to the universal love of pets, as well as the unique and often unexpected interactions between cats and dogs. Whether you're a cat person, a dog person, or both, funny cats and dogs videos are sure to provide a much-needed dose of laughter and joy.

Everyone knows all about different dog breeds. Dogs are much more associated with individual breeds than cats are, however. There are dozens of different breeds of cats, and each breed is different from one another, just like dogs. So how do you know what cat breed is right for you? You need to consider what you want out of a cat in your personal household before making a decision. Evaluate your living situation. The first thing you should do before settling on a cat breed is to evaluate your personal living situation. How many people are in the house? Are kids or other pets involved? Do you have room for a litter box? Do you spend a lot of time at home or a lot of time on the road? These questions are imperative. Some cat breeds are better with kids and pets than others. Some cat breeds are perfectly fine being left alone for hours or days at a time, while others are not. Large breed cats might feel cramped or obnoxious in a small apartment, outdoor or indoor, or both. Do you want an indoor cat or an outdoor cat? Indoor cats are very common, but they run the risk of running away or being scared of strangers or other cats if left isolated inside for too long. Outdoor cats are fun. But they have the risk of disappearing and not coming home, and you have to be prepared for that. Do you have property where a cat can safely roam nearby the house without disturbing the neighbors and the local wildlife? Outdoor cats also run the risk of getting parasites or getting into fights with other cats, which you will have to factor into your plan for medical treatment. Some cats are happy being led outside every once in a while, but are also content to spend most of their time inside, which might be a happy middle ground size and appearance. The next thing you want to consider is how you want your cat to look. Do you enjoy large cats with a lot of weight or lean and petite cats? Some cats are extremely large, like Maine Coon cats, which can weigh up to £10 or more. Other popular breeds like Siamese cats are much smaller. Large cats eat more, produce more waste, and if they have long hair they shed more. But large cats do have a striking appearance that appeals to many. Consider a cat's shedding. Speaking of shedding, cats shed a lot of hair. If you are allergic to cats or hate dust, you will definitely want to consider short hair cat breeds. There are short hair breeds and long hair breeds. Long haired cats have much thicker and longer fur. When these cats shed, they produce a higher volume of loose hair and therefore more allergies and associated particles. If you want a long haired cat, you should check to make sure no one in your household has allergies related to cats. You will also have to consider grooming. Even though they keep themselves well groomed, longhaired cats have the possibility of needing extra grooming due to their coats. Spend time with cats to gain some experience. If you don't really know how different cats act and behave, you should try and spend some time with cats to get more familiar with the species. There are various ways to do this. You can go to the local shelter and talk to the people there. Most shelters are more than happy to let you see the animals and spend time with them To see if you are a good fit. You can visit cat cafes. These new businesses allow you to sip coffee and play with cats. You can also ask your friends and family about their cats to see what they like or dislike about their particular cat. The more you know, the better informed you will be to make a decision that is right for you. Some cats are more independent than others. Some cats are very independent and can be left alone if you do not want to deal with them. Outdoor cats are very independent. Other breeds that are independent are British Shorthair, American Wirehair, and Persian cats. These cats can be left alone and will leave you alone if you work from home or are busy with children. Other cats, like Siamese cats, will need you to pet them, sit with them, and pay attention to them a lot more often. Needy cats that do not get enough stimulation can become a nuisance as they will bother you, meow, and sit on you when you don't particularly want them to. Playful versus chill. Some cats are extremely playful and energetic. These cats will need a lot of play time or a lot of appropriate stimulation. Energetic and playful cats will need teasers, cat trees, and plenty of exercises. If playful cats do not get the proper stimulation, they can take it out by shredding your furniture and your shoes, or even trying to play with you while you sleep. Energetic breeds include cats like Abyssinians, Bengals, and Savannah Cats. Chill cats will be more content to nap the day away than play. Chill cats include Persians, Siberians, and Russian Blue Cats. Pick a cat breed this video, kindly press the like button. Also, don't forget to subscribe with notifications on so that you don't miss out on videos like this. Thank you for watching.

Scientists have a clear moral obligation to clearly warn humanity of any catastrophic threat and to tell it like it is in November 2019, more than 11. 1000 scientists from 150 countries clearly and unequivocally declared that planet Earth is facing a climate emergency. CO2 levels are rising, the glaciers are melting, Antarctica is melting, the Ocean's getting hotter, more acidic, sea levels are rising, and so are extreme weather events. And yes, yeah, fossil fuel use is going up, like air travel, but so is per capita meat consumption. In fact, one of the solutions they offer to help the climate crisis is eating mostly plant based foods while reducing the global consumption of animal products. And what makes designing a sustainable diet so easy is that the same advice like eat less meat is good for both personal health, like reducing the risk of our number one killer, as well As for planetary health right the least healthy foods. Also caused the worst environmental impact. The foods with the most nutrition just so happen to be the foods that caused the lowest greenhouse gas emissions. So you get this win win effect. So let's put it all together. We are to redesign the global food system for human and planetary health, which is to say human health and future human health. What would it look like? Enter. The East Lancet Commission, the result of more than two years of collaboration between 37 experts from 16 countries suggesting a cut in total meat consumption down to like an ounce a day. It's like the weight of a single chicken nugget, right? All the while dramatically increasing or intakes of legumes, which are beans, split piece, chick peas and lentils, nuts, fruits and vegetables. Because we're not just in our climate crisis, but a health crisis, right? Unhealthy diets cause more death and disease than smoking, more than unsafe sex and alcohol, drug and tobacco use combined. But we can address both crises at the same time by increasing our consumption of whole plant foods and substantially reducing our consumption of animal source foods. Eating such a diet could save the lives of more than 10. Million people a year and may just help save the world. The Paris Agreement had set out a boundary condition. An aspirational goal for a carbon budget to help prevent catastrophic impacts and staying within the boundary for climate change can be achieved by consuming plantbased diets. And the personal benefits may be comparable with or even exceed the value of the environmental benefits. The healthcare benefits alone for a healthy global diet, predominately plant based diet, vegetarian or a vegan diet could exceed the price of the carbon saved. We're talking up to $30 trillion a year saved from the health benefits alone. Now, if the health of yourself, the planet, and your own children doesn't quite motivate you, consider that you may also be facing threats to the global beer supply. And healthy diets don't just reduce greenhouse gas emissions. Since livestock production is the single largest driver of habitat loss, reducing meat consumption is also the key to biodiversity conservation. Ideally perhaps, reducing demand for animalbased foods by increasing the proportion of plantbased foods up to like 90% of the diet. Livestock production is also a leading cause of a soil loss, water and nutrient pollution. Yet it appears to be a blind spot in water policy. Despite the fact that animal products form the single most important factor in humanity's water footprint, water managers never seem to talk about meat and dairy. But it's not just animal products. I mean, yes at least. 80% of the deforestation in the Amazon is to raise cattle and grow feed crops like soybeans to export to other farm animals, but also to make vegetable oil, most of which is from palm and soy. Both crops have been expanding, resulting in massive deforestation. It just seems kind of particularly egregious if that deforestation takes place for the sake of junk food. Not everyone agrees we should be moving to healthier diets though. The World Health Organization actually pulled out of the E Atlantic Commission because of their promotion of a global move to more plant based foods. See, if we focused on promoting predominant plant based foods and excluding foods deemed unhealthy, including meat and other animal based foods, such a diet could. Yeah, save 10 million lives a year, $30 trillion, and help save the entire planet. But could lead to the loss of jobs linked to animal husbandry and the production of junk.

Global meat production has skyrocketed over the last half century, with pork and poultry meat now exceeding 100 megatons a year, 100 million tons, and this growing demand is unsustainable. The reduction of animal products is arguably one of the most impactful ways in which individual consumers can alter their diets to possibly impact individual and societal wellbeing, and there's a definitely growing interest in plant based diets and meat reduction. But even just something like Meatless Mondays requires dietary change and sadly neither sustainability or health approaches are likely to work with those who love their meat. But swapping and plant based meat substitutes may help. Help kind of disrupt the negativity about reducing meat, but for hardcore meat eaters, it's got to taste like it and look like it. It's interesting. The more people consume meat substitutes, the less likely they are to care that has a similar taste, texture, appearance or smell of meat. But to appeal to those who really need them are the meteor the better. This has certainly been accomplished with the spate of new products on the market with all studies of grain that they're. Healthier for the planet, but what about healthier for us? Comparing labels of the burgers and looking at for the worst components of the food supply, trans fats, saturated fats, sodium, and cholesterol, the plantbased burgers win hands down when it comes to trans fat and cholesterol. We all know trans fats is a serious potential risk factor for cardiovascular disease, cancer, and diabetes, but it's also been recently associated with. Symptoms of depression Lower testosterone in men even just like 1% of calories and dementia. Higher levels of trans fats in the bloods associated with up to 50%. High risk of developing dementia, including Alzheimer's. Now that partially hydrogenated oils have been phased out of the food supply, the only major source of trans fats left will be from animal products. What's the. Tolerable Upper Daily Intake Level for trans Fats and Upper Limit was not set for trans fat by the Institute of Medicine because any incremental increase in trans fat intake increases the risk of heart disease, the number one killer of men and women, as in any intake above 0. Because trans fatty acids are unavoidable in diets that can take meat or dairy, consuming 0 trans fat would require significant changes in patterns of dietary intake. One of the authors to the report from Harvard's nutrition department, offered a memorable explanation for why the Institute of Medicine panel didn't you know, cap it at zero. We can't tell people to stop eating all meat and all dairy products, he said. Well, we could tell people to become vegetarians yet, and we were truly basing this only on science. We would, but it is a bit extreme. Wouldn't want scientists to base anything on science, now would we know. But anyways, that's a big advantage. And of course, no hormones, no antibiotics, hasn't been, you know, designated as probably cancer causing by the World Health Organization and on and on. Now I'm not happy with the added salt, which is about 1/4 the American Heart Association is 1500 milligram daily upper sodium limit or the saturated fat. Thanks to added coconut oil, but these two seem to be outliers. I'm in the largest study of the nutritional value of plant based meats today. Saturated fat levels of similar products only average about two grams per serving, much better than the animal based equivalents. Sodium remains a problem throughout the sector, though like nearly any other processed food out there. How processed are. These products, well, I mean, if you look at the fiber content, for example, yes, I mean to see any fiber in a burger, that's a good thing. But I mean compare that to a whole food, right? If you ate the same amount of protein from yellow peas, for example, the primary plant protein, and beyond burgers, there'd be almost no saturated fat and sodium and a whopping 20 grams of fiber. So yes, you know, processing plants in a processing plant can eliminate 90% of the fiber, but processing plants through animals eliminates. 100% of the fiber. So of course, as the chair of Harvard's nutrition department put it, you know, nutrition policies and dietary guidelines should continue to emphasize a diet rich in whole plant foods, such as nuts, seeds and legumes or pulses, which are rich in protein and many other nutrients but require little industrial processing. But we shouldn't let the perfect be the enemy of the good. Not everyone can go all you know kale and quinoa overnight. The choice on the Burger King menu is in between this and this, right? But between this and this, and in that case, it's a number brainer. https://www.majalatsayidaty.com/ https://www.infocancers.com/

Hello, it's Samantha again and I'm here with another awkward introduction. If you clicked on this video, you probably read the title and decided you want to watch it. I hate me. I'm going to be explaining all about a C chemotherapy. I have no idea what that stands for. I'm going to Google it real quick. It's not air conditioning. It's not alternating current cyclo. Close. How do you say this? I'm gonna Google how to say this. Cyclophosporide sag Sag, cyclosite, cyclophospide from my cyclophosporide cyclophosphamide. Cyclophospor A/C stands for adriamycin and cyclophosphamide. I'm going to be explaining what happens when you get it. The side effects that I had, yeah, and probably some other stuff that I can't think of right now. So I already finished my A/C chemotherapy. I had four rounds of it, so I don't have a full on blog, but I've got lots of videos and pictures of everything. April 5th was my first treatment and I did it every other week from then. It's basically a typical day for me arrive at the Cancer Center. It took me like probably a full month of going there. To stop laughing at the name because it just sounds really serious. I would get my stylish bracelet that has my name and my birthday on it. I would wait in the waiting room until they were ready. Then I would go back by myself and they would access my port. It's just a medical device that is in my body. They did surgery to put it in so it's under the skin and they stick it. To give me my chemotherapy. It's just so that they don't have to give me an IV and my hand or my arm every time because the chemotherapy would tear up my veins in my arm. So if it goes through the port, it gets directly pumped out through bigger veins and stuff that can handle it. But what they do when I go back there is that they just take some blood so that they can send it to the lab and they can check and make sure that everything looks OK Then I go away in the waiting room a little bit longer and then I go meet with my. Oncologist Every week the nurse asks me a bunch of questions about how I'm doing on the chemo and my side effects and everything. And then she asked my stress level. And I always say it's great because I think that's a dumb question. And then they just measure the bump. In my breast and check to see if it's gotten any smaller. Mine did get smaller. It started out 3.5 by 3.5 centimeters. By the 3rd treatment I think it was 2.5 by 2.5. So after my checkup I go and I find a place to sit. Usually like to try to find a heated chair because I get cold easily they have to do a few things before they start giving me the chemo. In the checkup they check my height and weight. I don't really know why they need to keep checking my height because I'm done growing. The amount of chemo that they gave me is very dependent on my height and weight. For instance, on the first day that I went, I had a lot of extra weight leftover from my egg retrieval surgery for the adriamycin. They had two big syringes of it. They had one tiny little syringe. They all were joking about how it was really funny that there was like this tiny little syringe with a little bit of medicine. Then the second day I came in for chemo. I had lost £5 by then because I was just going back to my regular weight and that extra little syringe was gone. They didn't use it at all, so it really doesn't matter how much they give you based on your weight. Even like £2.00 can make a small difference. Anyway, I go and sit in the chair and before they can even give me the chemo, they have to give me nausea medicine. Some of the nausea medicine is pills. And some of the nausea medicine, they pump directly in through my port, they get the medicine from the pharmacy. They start with the Adriamycin. It is very red. They can't just put that on the little machine to pump directly in through my port because it's very important that someone, a nurse, is there to watch it being put in and it is important for them to keep checking as they're putting it in if there is blood coming back. So they push the red medicine in and then they pull back and they make sure that they're still getting blood return. It can cause severe, severe burning if it doesn't go directly into the veins. The nurse also needs to wear a huge cover and protect themselves while they're injecting this into me because if any of it gets on them, they saw that it caused bladder cancer, so. This is not normal medicine. It's a little bit scary. They're basically just putting poison into you. Also, because of that red color, it afterwards turns all your bodily fluids red. Your urine will be red, basically bright red the very first time you'd pee. And then after that it starts fading a bit away, gets like orange, and then this is really gross. Starts turning back to the regular color as the week goes on. It's supposed to do like, all your fluids. So somebody said that they tried to cry to see if their tears would be red. I don't know if they were successful. I don't know. So if you're doing this, that could be something to try. OK, so after they drew my sin, they have to do the other one. And I'm not going to try to pronounce it again. And that one can just go straight up on the machine. I guess they do it over 30 minutes usually. I only did it over 30 minutes. The first time I went in because that one can cause your nose to start burning. There's probably the last 10 minutes of getting the medicine that I actually noticed it and my nose just kind of started burning like in the inside. After a while, it started like going up into my head and I like felt like my brain was burning, if that makes sense. It just caused a headache. By the end I only had like 2 more minutes of it. I was like, all right, just keep going, I'll make it through. What helped me was instead of going over 30 minutes, the next time they did it over a full hour, I still every single time felt the burning. But it was only in that like last five or 10 minutes. So I feel it so usually just pushed through and it was never as bad as the first time. It never caused the full on headache, the headache and everything completely went away after 20 minutes. So it's not like it causes permanent. Damage or anything. While they are giving the second infusion, they attach a Neulasta patch. They put it on my arm, they can put it in your stomach or basically wherever you're comfortable. After they put it on, it starts beeping. It tells you that it's going to stick you, it makes this a whacking noise and then it injects the little tube thingy, I guess.

Metastasis is the leading cause of cancer related death. Cancer kills because cancer spreads. For example, the five year survival rate for women with localized breast cancer is nearly 99%, but that drops to just 27% in those with. Metastasize cancer. Yet our ability to effectively treat metastatic disease has not changed significantly in the past few decades. You know things are getting desperate when there are papers like this targeting metastasis with snake toxins. Now we do have built in defenses, natural killer cells that roam the body, killing off budding tumors, and I have videos on boosting natural killer cell activity. But as I explained in the last video, there's a fat receptor called CD36. It appears to be essential for cancer cells to spread, and these cancer cells respond to dietary fat intake, but not all fat. CD36 is up regulated by palmetic acid as much as a 50 fold increase within 12 hours. Palmetic acid is a saturated fat found in junk food made from palm oil, but it's most concentrated in meat and dairy. This may explain why if you look at dietary fat and breast cancer mortality, there was no difference in risk of breast cancer specific death for women in the highest versus lowest category of total fat intake. But you're about 50% more likely to die of breast cancer. Feed a lot of saturated fat. The systematic review of metaanalysis conclude that saturated fat intake negatively impacts upon breast cancer survival. This may explain why intake of high fat dairy but not low fat dairy was related to a high risk of mortality after a breast cancer diagnosis if it was the dairy. Protein. Like caseine. That was a problem. The skim milk might be even worse, but no, it was the saturated butter fat, maybe because it triggered the CD36 induced cancer spreading mechanism. Women who consumed one or more servings per day of high fat dairy had about a 50% high risk of dying from breast cancer. We see the same thing with dairy intake in relation to prostate cancer survival and drinking high fat milk compared to increase the risk of dying from prostate cancer by as much as 600% in patients with localized prostate cancer. But low fat milk was not associated with such an increase in risk, so it seemed to be the animal fat rather than the animal protein. And this is consistent with what Harvard researchers found in the United States. More evidence that the fat receptor CD36 is involved is that the risk of colorectal cancer from meat consumption increased from just doubling risk to octopling risk, multiplying the odds of getting cancer eightfold for those who carry a specific type of CD36 gene. So is it time to give breast cancer patients, for example, a prescription for a low fat diet? A cancer diagnosis is a. Teachable moment, if there ever was one, to motivate people to make changes to their lifestyle. But provision of evidence based guidelines is essential and you don't know until you put it to the test. A randomized perspective, multicenter clinical trial to test the effect of a dietary intervention designed to reduce fat intake. In women with resected early stage breast cancer, meaning the women had their breast cancer surgically removed and we're praying it doesn't come back, the dietary intervention group dropped their fat intake from about 30% of calories down to 20% of calories, dropped saturated fat intake about 40% and maintain that 40% lower intake after one year, three years, five years and. After approximately 5 years of fall, the women in the dietary intervention group had a 24% lower risk of relapse, a 24% lower risk of the cancer coming back. That was the WINDS study, the Women's Intervention Nutrition study. Then there was the Woman's Health Initiative study, where again, women were randomized to drop their fat intake down to about 20% of calories. And again, those in the dietary intervention group experienced increased breast cancer survival, meaning a dietary change may be able to influence breast cancer outcomes. And not only was breast cancer survival significantly greater, the women also experienced a reduction in heart disease and a reduction in diabetes as a little side bonus.

Our lifetime risk of developing an invasive cancer, not like some superficial skin cancer like ductal carcinoma of the breast, but serious cancer is about 40%. Two in five of us are going to get a cancer diagnosis in our lifetimes. What can we do? To reduce our risk, only about 5% of cancers are caused by problem genes we inherited from our parents. The other 95% are caused by mutations in our DNA we acquire in our lifetimes, for example based on a genetic analysis of lung cancer smokers. May acquire an average of 1D NA mutation for every 15 cigarettes smoked. Smoking is bad, but the number one cause of these mutations is our diet, and that's not even including the cancers attributed to obesity. I've got tons of videos on dietary approaches to prevent cancer, but what if you already have it well meaning? Professional sometimes counsel cancer patients that. Eat whatever you want. Given the time constraints, that doctors phase may be understandable that the treating oncologist the the treating cancer doctor may be reluctant to engage in a conversation about nutrition. But given the critical role that diet may play, perhaps it should be a critical part of their job to be able to answer patients questions about nutrition before and after cancer treatment and not default to the unhelpful. It doesn't really matter. Eat whatever you want which. May not be in the best interest of the patient. The official recommendation of the American Institute of Cancer Research, a limiting authority on dying cancer, is that those with cancer should follow the same diet that helps prevent cancer from taking root in the 1st place. That means more whole grains, vegetables, fruits and beans, while limiting fast food, processed food, meat, soda and alcohol. Similar recommendations have been put forth by other cancer authorities. More fruit. Vegetables, whole grains and beans, and less salt, sugar, meat and alcohol. Cancer survivors adhering to these guidelines do seem to live significantly longer, or at least older female cancer survivors. The only. Group in which it's been looked at so far there that there are certain foods that may be beneficial in cancer care including beans, berries, cruciferous vegetables, flaxseed, garlic, green tea, tomatoes and others. But you know emphasize it's not about a single magic bullet food or component, but the combination of foods in a predominantly plant based diet. Here's how some popular diets used by cancer patients stack up. The so-called alkaline diet gets high marks for being vegetable focused and encouraging people to cut down on animal foods. The. Keto diet does the worst though. They get points for keeping people away from refined grains, alcohol and soft drinks. Macrobiotic diets you really kind of win the day being closest to a whole food plant based I centered around whole grains, vegetables and beans though may not be advising enough fruit. Paleo diets are a mixed bag with insufficient whole grains and beans and too much meat, and the vegan diet starts out strong, but doesn't necessarily preclude all manner of vegan junk food. Have any of these diets been put to the test? I've done a video on the abject failure of the keto diet. The alkaline diet was tried on 11 lung cancer patients. They lived an average of 20 1/2 months. Which is about 40% longer than most patients have historically lived, but there was no direct control group. The only diet proven in our randomized control trial to reverse the progression of cancer was Doctor Dean Orange's Whole food plantbased lifestyle program, which I've covered before most randomized controlled trials to date on diet and cancer. Are like this right? Feasibility studies? Just to see if, like, you can even get cancer patients to eat healthier. Otherwise, what's the point of even running the study here? They did find they could get patients with head and neck cancer to ramp up green, leafy and cruciferous vegetable intake up to 9 cups a week. So it's at least something you could test. We don't yet have outcomes data, but why wait? What's the downside of trying to eat healthier? It may. Even Save Your life, another way cardiovascular disease competes with breast cancer as the leading cause of death for older women diagnosed with breast cancer. Researchers fall more than 60,000 women diagnosed with breast cancer over the age of 65 for an average of nine years, by which time half had died. And the number one cause of death was actually cardiovascular disease edging out the breast cancer. And so choosing a healthy diet centered around whole planned foods, the only diet ever proven to reverse heart disease in the majority of patients, may save your life, whether you have cancer or not.

In a recent interview with Dr. Leonard Gamella at the Kimmel Center at Jefferson, he discussed the final results of the phase three COU 3O2 study that were just presented at the ESMO meeting in Madrid. This was the longterm study of the Aberraterone 3O2 trial that looked at the use of Aberraterone before docetaxel chemotherapy and men with metastatic castrate resistant prostate cancer. And this long term data showed convincingly that there was a benefit to using abiraterone with Prednisone in this patient population. What was very important about the study is that the final analysis was about a 34.7 month survival in the treated group versus about 30 point three months in the placebo. Arm of the study which was just Prednisone. So this convincingly shown that there is a survival advantage to using abiraterone in this patient population. In this particular Cougar 302 study there was a there was a crossover about 66% or about the 2/3 of the men. In the the treatment arm in about 80% of the men in the placebo arm ended up crossing over to the Aberraterone arm. So they ended up getting actual treatment. What was very important about it is that regardless of whether you were in the treatment arm or the placebo arm, the fact that the subsequent treatment really did not make a difference, the results held true that the use of aberraterone did extend to survival even in those that were in the crossover arm. Concerning starting the the use of these agents for a metastatic history resistant prostate cancer, we probably like to start them within a month or two. Sometimes you know three or four months can make a difference. So you want to try to get these agents on board as early as possible. The take home messages of using the abiraterone in in these patients is that really a lot of it has to do with quality of life and survival. Some of the benefits in this particular trial of using abiraterone really related to the delayed use of opioids sometimes as long as he possibly is 12 months or longer, meaning that men are not having discomfort, they're not suffering a lot of morbidity from prostate cancer and their quality of life is is greatly improved. So it's good that we now have these agents that we can use in patients that are relatively. Well tolerated side effects profiles are really very, very limited. You do have to monitor liver function tests and some basic chemistry parameters, but overall the drugs are very well tolerated and give a good quality of life to men who have an otherwise very difficult situation.

Hi guys, welcome to Empower and my name is Caroline Porter Thomas. Thank you so much, as usual, for watching my YouTube channel. So today I have something very special for you. I have an interview with a wonderful woman. Her name is Camille and I met Camille when I was at a fundraiser for University of Miami, which is where my husband works. She was the main speaker and she shared her story and it touched me so much as a nurse and somebody in the healthcare fields. That I said to myself, I absolutely have to ask her if she will come on and do a video for you guys because her story is so inspiring. You know, working in healthcare, we see people at their worst, you know, at their lowest, their darkest days a lot of times, You know, we never really get to see the people that heal. And there's a lot of people that do overcome the almost impossible or the impossible, you know, I understand it. They got better. And they never want to step foot in the hospital again. And I probably, I probably wouldn't either. I just, I wanted to share her message with you guys, nurses, nursing students. Also, I know of course that there's a lot of other people that watch this channel. You could be a patient yourself, your mother could be your father, brother, sister, anybody. So this is a message of hope. There's always hope. And hope. Is free. Hope is free, and so without any further ado, I'm going to let Camille take the stage that she truly deserves and you're going to love this video. Prior to. Being diagnosed with cancer, I pretty much was busy. I like to ride my bike, I like to go to the beach, I like to walk my dogs. I spend a lot of time outside gardening, Just kind of living life. Nothing out of the ordinary, just a really a normal person. Just living my life, working, being around my family, enjoying my home time of course, and that's kind of it. The day that I went to the emergency room because I was having pain, they did a CT scan and they came back right away and told me there are spots on your pancreas, your liver and your lungs, which was terrifying. I was very, very frightened. My mother died of pancreatic cancer when I was 24. She was 64. So my immediate thought was. I was in some really bad trouble. I was very scared. I think the first thing I did was call my oldest brother on the phone. I just told him things are not looking good. I'm sick and I didn't want to talk about it anymore because I didn't want to cry. So I said I have to hang up now because I don't want to cry. And then I was admitted. I stayed there for seven days. I had a biopsy and I kind of was a little calm. Even my daughter kept thinking you're really calm and I. I sort of stayed calm until the day that everyone came in my room at one time. And I knew that that wasn't going to be good news because it was like the psychiatrist came in a doctor. He wasn't the oncologist, he was just the regular MD and a whole bunch of nurses. And I knew that wasn't going to be a good news. After I was told for certain that I had cancer, that you know when they did the liver biopsy and they knew it was really cancer and he told me it is cancer, I was scared and I. Cried and I asked for Valium. I wanted to calm down. That was the first thing I did. I went to sleep and then I woke up and when I woke up I had this overall weird calmness again and kind of was taking everything in just day-to-day, thinking about what chemo would be like. Not really wanting to do it, but I just sort of. I was scared, panicked, but something way down. I just, I didn't want to think I was going to die. But everybody else around me seemed to think that, but I refused to think it so. Chemo. There was a lot of tough days because I went through eight months of very, very aggressive treatment that made it impossible to eat. It made it impossible to sleep. It was just really horrendous. It caused a lot of side effects. It caused me to have neuropathy where I can't feel my hands or feet, caused a lot of side effects. But eating was a chore. It was really a chore to eat. I think I got down to 90 pounds. I was very thin, weak. I think probably my worst day would have been I had a splenetic embolism, which they go in through your groin with some wires up into your spleen and they closed down your spleen to save your blood supply and your body longer because I kept getting issues with low platelets which would make me not be able to get treatment. So that was the worst day because you have to be awake and you have to. It took five hours and I was laying on a table. Flat for five hours and awake so and you see the the TV screen and the wire going through your body and you'll be told to hold your breath at times and things like that. So that was probably the worst day and I just wanted it to be over and go home and and and I went to recovery. They gave me some medicine there to like keep me calm and then I woke up and got out of there and went home. So that was a pretty bad day. The first treatment I had was probably the worst and scariest because as you're sitting there, you're with these nurses and you are assigned a nurse, which was amazing. She was with me throughout that whole day and that was a very long day. That was probably a 12 hour day. I used to get chemotherapy for about 10 hours and then I would get a pump attached to my chest and I would go home with a pump. That remained for 46 hours with chemotherapy, so. That was a very challenging time. It was exhausting it. The first day was very scary because you look at those bags and you're like, Oh my God, is that the chemo? Is that the chemo? And the first, you know, you think all of the the chemo is going to go in your body and you're going to have this little, Oh my God, chemo is in my body. And it's really, it's not like that. And you don't get sick and and and they give you so many premedicines these days that you really don't feel that. You feel tired. You feel a lot. Tired. You have some issues eating, but it did save my life and it worked. So I don't know. I can't say it was good, but I can't say it was bad because it was just something I had to go through and I'm happy not to be on it anymore.

A countless number of patients suffer every day from agonizing pain that is caused by various types of cancers. A conventional treatment option that has been used to treat cancer patients for decades like chemotherapy, helped treat cancer disease but leaves patient with extensive damage from its side effects. With advancement in healthcare technology, a breakthrough has been made in the field of Cancer Research. The discovery of targeted therapy in recent years is used to treat cancer cells by targeting or attacking specific genes or proteins that are involved in the growth of cancer cells without damaging other parts of the body. In target therapy, this is a medication the point of mutation that the cause of cancer. So if we are use the target therapy, we can stop the cancer with the more efficacy. But the less side effect. Immunotherapy relies on the function of the patient's immune system. Typically, the immune system monitors and destroys foreign substances such as germs and cancer cells. Sometimes the immune system fails to detect those substances. Immunotherapies then used to help boost. The effectiveness of the patient's immune system so it can detect and fight against cancer. The immunotherapy is the medication that enhance patient immunity to cure the cancer by the patient immunity. So the side effect is less than all chemotherapy but the efficacy is good and sustains ability. The treatment method depends on the several factors such as the location of the cancer cells. The severity of the disease and the type of cancer the patient has. For example, in liver cancer patients, targeted therapy can be used along with other treatment methods. However, cancer cells or tumors must be tested before the treatment option is chosen. The stage of cancer the patient has at the moment also plays a crucial role in receiving proper treatment. Each options of treatment cancer. Including target therapy, immunotherapy or news chemotherapy depend on type of cancer and stage of cancer. For example in lung cancer we are use target therapy instead four of cancer but immunotherapy we use instead three of cancer. So it very hard to say what stage of cancer or what type of cancer can use the new option of treatment. So I suggest you to come to see a doctor for horn cells you can use the new therapy or not. Our treatments can use the same time together depend on the disease to some disease can use together for example in the hepatol, cellular carcinoma or liver cancer we can use together with. Immunotherapy and target therapy, but in some kinds of cancer, for example lung cancer, we individually use target therapy early or immunotherapy early or combination use immunotherapy with chemotherapy for. Increase efficacy of treatment. Targeted therapy precisely targets cancer cells and does not affect the nearby region, making it less harmful to the healthy cells when compared to chemotherapy. The key benefit of immunotherapy, on the other hand, is that the chance of being completely cured is relatively high when the treatment begins from an early stage. Among the innovative treatments, the side effect difference depend on the type of treatment. In target therapy, the side effect is not too much, OK, but we have the specific side effect depend on the target therapy. For example, the target therapy that used in lung cancer, the patient maybe have a last or hepatitis that we are closed for up by doctors. And immunotherapy, the side effect is less than chemotherapy, but because the immunotherapy enhance the patient immunity, so some people may be inflammation in the body, for example, dermatitis, hepatitis or colitis that this symptom the doctor will be secularly and caused for the app in the part of chemotherapy the side effects depend on. The medication that the doctor will be informed you before start treatments and call follow up about the side effects to you. This innovative approach to cancer could make a significant difference for cancer patients today. Technology and research in the medical field are rapidly growing, making new treatment options like targeted therapy and immunotherapy, as well as the new type of chemotherapy. Highly effective with satisfying response rate that also stress the possibility of extending patients lives. Cancer is an alarming disease, but it's crucial to understand and it can be prevented if it's detected early. Undergoing cancer screening tests at least once a year could reduce the chance of developing the disease. Wettany victory for life. Leave us a comment below if you have any questions, and please hit the subscribe button to stay updated with our healthcare videos. https://tafsir-almanam.blogspot.com/

Cancer affects us all, and it's pretty likely you know someone who has had this disease. In fact, one in three people will be diagnosed with cancer in their lifetime. Cancer occurs when our own cells divide uncontrollably, avoid the many protections against abnormal cells, grow into large tumors, and spread uncontrollably. But how does that happen, and why? That's a great question that has puzzled scientists for hundreds of years. In fact, it turns out that some cancers are driven by cells called cancer stem cells. Cancer stem cells are different from normal, healthy stem cells since they divide and survive outside of the body's control. These cells give rise to the many cancer cells that divide uncontrollably to form a tumor. That spreads unchecked throughout the body. This happens in breast cancer. But how do these cancer cells divide seemingly unchecked? Part of the answer involves the immune system. When we think of the immune system, we think of its role in keeping us healthy and killing disease causing pathogens that enter our body, such as bacteria or viruses. But the immune system is also in charge of killing any cell that may start to misbehave in an attempt to prevent cancer. However, some cancer cells acquire changes in their DNA sequence and begin to produce high levels of proteins that render these cells invisible to the immune system. One such invisibility cloak is the protein CD47. Which helps give cancer cells and cancer stem cells the power to cloak themselves from the immune system's defenses. Normally you have special immune cells known as macrophages, literally big eating cells that physically gobble up bacteria or abnormal cells such as cancer cells. However, invisibility cloak CD47 renders cancer cells invisible to macrophages, allowing the cancer cell to divide and supervise and eventually form a tumor. To repeal the invisibility cloak and make cancer cells vulnerable to the immune system, scientists at Stanford University devised an ingenious strategy. They designed an antibody that can bind it to CD47 and disrupt the invisibility cloak. Antibodies bind and identify material to be destroyed. So what did these scientists have in mind with this CD47 antibody? Here is their idea. When the CD47 antibody is present, it would bind to CD47 on the surface of cancer cells, removing the invisibility cloak and alerting the immune system that something is wrong with the CD47 expressing cancer cell and that it should be destroyed. Studies in Petri dishes and mice suggest that this actually happens. When the CD47 antibody is present, most cancer cells are destroyed by macrophages, those big eating immune cells. When the CD47 cell is eaten by macrophages, they digest the cell into smaller components, which activate the immune system even more. This amplified immune response creates a stronger attack against the cancer cells, which could help destroy the remaining ones. Using this approach, the scientists would like to harness the body's own defense mechanism to kill the cancer stem cells, which often persist after chemotherapy and radiation and may be responsible for cancer relapse. By helping the immune system identify what is cancer and removing the cells in visibility cloak, we can make cancer stem cells vulnerable to the body's own defense system thanks to funding from Proposition 71. Scientists at Stanford University are now testing the CD47 antibody for safety and potential efficacy in a human clinical trial of breast cancer patients. This approach could be applied to many other cancers, such as certain types of colon cancers and leukemias. As scientists and patient advocates build on the progress that Proposition 71 funding has enabled, we must keep the momentum going, understanding that there's still much work to be done. We must remember that human trials will celebrate successes, but barriers will surface along with complications and challenges, so patience and understanding of the scientific discovery process are essential.

So the tumor microenvironment of pancreatic tumors tends to be what we describe as nutrient poor. So the cancer cells have less access than many normal cells to the sugars and other important nutrients that help cells proliferate. And so because of that, pancreatic cancer cells have developed these sort of alternative adaptive mechanisms. That allow them to take up nutrients differently from their environment and also use them differently once they get into the cell. One of these processes is autophagy which some people might have heard of as self eating. So this is when the cell actually breaks down things that are already inside the cell, whether they they're organelles or proteins and uses the fuel that they generate from those to help them proliferate. And sort of where we went with our story is that we found this. Pathway called mytopagy. So this is a form of autophagy where specifically the mitochondria are broken down and then recycled for their different parts. We found that this pathway seems to be one of the ways that pancreatic cancer cells adapts to those low nutrient environments. So one of the things that we hope to do now is sort of see how we can harness our understanding of this altered. Sugar metabolism and also changes in mitochondrial dynamics into a therapeutic strategy for patients.

My name's Erica and in January 2012 I was diagnosed with pancreatic cancer. It's since metastasized into my liver and also a lymph node behind my pancreas. Every second person will develop cancer in their lifetime. Pancreatic cancer is one of the most aggressive. One of the things that hasn't changed over the last 30 years is the poor prognosis for people diagnosed with pancreatic cancer. The five year survival rate is only 5%, which is a pretty gloomy outcome for those of us who've been diagnosed with it. Pancreatic cancer is particularly lethal because by the time it is detected, it's already advanced. That's partly because the pancreas sits in the abdomen very deep and doesn't cause much in the way of symptoms as the cancer develops. And these symptoms are easily confused with general aches and pains. I think that's one of the hard things about pancreatic cancer is that the symptoms are vague and that they're easily attributed to something else. They're like stress, air, whole bowel syndrome, cancers. Form when normal cells in our body start to divide out of control over time. The growing cancer mass destroys nearby healthy cells. Many of the triggers that cause a normal cell to start to divide out of control are found inside the cell in our DNA. Our DNA encodes the genetic information needed to assemble molecules such as proteins, which are the building blocks of our bodies. DNA is under constant attack from natural chemicals, environmental toxins and background radiation. Here we can see some toxins, shown as bright particles interacting with the DNA and causing the DNA strand to break or causing mutation in the genetic code. Cancer arises through an accumulation of damage to the DNA. In a normal, healthy cell, molecules called P53 are activated following damage to the DN. AP53 is part of our natural defense mechanism against cancer. By binding to critical parts of the DN, AP53 molecules help guide the cell's response to the DNA damage. Sometimes the defense mechanism itself can become damaged and P53 is mutated in over half of human cancers. This means that P53 cannot bind to DNA, and without P 53 / a long period of time, cells can accumulate more and more DNA damage, increasing the risk of cancer. Mutated P53 is just one of thousands of molecular mistakes that can occur in cancer, and modern DNA sequencing and other new technologies can now enable these molecular mistakes to be detected in individual cancers. This is important because. Even though cancers may look the same under the microscope, we now know they can behave very differently depending on their genetic makeup. What's really exciting is that two different types of cancers that look different under the microscope can share some common genetic characteristics. This means that drugs that were developed for one cancer could also be used to treat the other cancer. I know that they're working on more personalized treatment. It seems that pancreatic cancer's been one of those ones that been a bit neglected in the past, but there's also a difficult one to deal with. I don't define myself by pancreatic cancer. That's a big thing for me. I I I feel affronted. When the press continues to say that somebody lost their battle with cancer, that's. We're all going to die. Does that mean we're all going to die? Losers in the press's eyes? I don't want to focus on battling cancer. I want to focus on living.

https://www.infocancers.com/ For decades, Japan is at the longest life expectancy in the world, while spending just a fraction on healthcare compared to other high income countries. This longevity has been attributed in part to Japanese dietary patterns, which are thought to have contributed to their comparatively low rates of coronary artery disease. Japan has historically had amongst the lowest rates of colon cancer, breast cancer, ovarian cancer, prostate cancer, bladder and blood cancers. Japan, however, has among the highest rates of stomach cancer. Yes, Japanese men may have had seven times less prostate cancer than Americans, but got six times more stomach cancer. Is there some Achilles heel in the Japanese diet? One of the first theories proposed in the 1970s was that it was the talc used to Polish white rice to give it a glossy Sheen. That was the case with ovarian cancer, which led to billions in damages against Johnson and Johnson's baby powder, as I detailed in the previous video. But that did not appear to be the case with stomach cancer. Is it just genetics? No. Studies on Japanese migrants show that as they and their children Westernize their diets and lifestyles, their stomach cancer rates drop accordingly. Well, the most well established risk factor for stomach cancer is H pylori, bacteria that infects the lining of the stomach and causes the chronic inflammation that can lead to cancer. H pylori infection is considered a Group 1 carcinogen, indicating our highest level of certainty that it indeed causes cancer. Korea and Japan have the highest rates of stomach cancer and among the highest incidence of H pylori infection. Case closed then, right? The mystery seemed to have been solved. But then came the African enigma. Countries such as Nigeria had even more H pylori, but only a fraction of Japan's stomach cancer rates. Then came the Indian enigma. H pylori is twice as prevalent in India than Japan. Yet Indians get 10 times less stomach cancer. Obviously, H pylori alone can't explain Japan's epidemic. The most cases of stomach cancer are thought caused by H pylori. Most people with H pylori don't get cancer. H pylori is one of the most common human infections. It's been estimated that half of the world's adult population is infected with H pylori, yet half of us don't get stomach cancer. There must be some kind of cofactor in countries like Korea and Japan that explains their elevated cancer rates. The inflammation caused by H pylori may just set the stage for cancer formation, increasing the susceptibility of the stomach lining to dietary carcinogens. But what's so carcinogenic about Korean and Japanese diets? Studies that have compared the dietary components of different Asian populations with similar H pylori rates but dramatically different stomach cancer rates. Have suggested preserved salted foods, both fish and vegetables, as the culprits. Fresh vegetables and fruits, on the other hand, were associated with an 85% reduction in stomach cancer rods, whereas consumption of fresh fish doesn't appear associated with stomach cancer either way. Or a view of 60 Studies found that the assumption of pickled foods was associated with significantly higher rates of stomach cancer, the more so in Korea than Japan, perhaps because per capita Korean consumption of salt fermented vegetables like kimchi is five day times greater. You can't know for sure, though, until you put it to the test. Pickled vegetable extracts can cause DNA damage in cells in a Petri dish, but what about in people? Researchers in Vancouver fed people 30 ounces of Fuku Genzuke assorted vegetables pickled in soy sauce or pickled cucumbers over three day. Biopsies taken from their stomach lining before the experiment started were normal, as were the biopsies taken after eating fresh carrots or cucumbers. But after just a few days of consuming pickled vegetables, moderate to severe tissue abnormalities were found suggestive of stomach irritation. The consumption of non fermented soy foods such as tofu, edamame and soy milk is linked to a lower risk of stomach cancer in Japan, whereas no association was found with fermented soy foods, even highly salted miso. The protection afforded by soy foods was attributed to the antiinflammatory and antioxidant effects of the isoflavone compounds and soybeans. Salt itself isn't considered a direct carcinogen, but it may damage the stomach lining. Thin the protective mucus layer, enhance H pylori colonization, enhance the penetration of carcinogens, and enhance the formation of carcinogens. Even moderately high salt intake is associated with significantly increased risk of stomach cancer, though in Japan this effect may be limited to those already suffering from H pylori induced inflammation. H pylori is typically treated with a cocktail of multiple antibiotics. Is there any way to eradicate it naturally through diet? We'll find out next.