Kevin McKernan: mechanisms of action behind ivermectin & fenbendazole treating turbo cancers

Kevin McKernan describes potential mechanisms of action behind ivermectin and fenbendazole treating turbo cancers.

"The combo of those two drugs makes total sense to me for fighting anything that might be negatively associated with the Pfizer vaccine."

"I was just fumbling through ChatGPT one day when I was reading Dr. [William] Makis' work realizing he's having a lot of success with ivermectin on these turbo cancers," McKernan (@Kevin_McKernan), Chief Scientific Officer and Founder of Medicinal Genomics, as well as former R&D lead of the Human Genome Project, tells John Davidson (@BrokenTruthTV). "Is there any connection between ivermectin and and P53? Is there any connection between it andSV40? And it comes back with a handful of papers saying, 'Yes. Absolutely."

For reference, P53 is a tumor suppressor protein that regulates cell division and helps prevent cancer by repairing damaged DNA or triggering apoptosis. SV40, in this context, is a reference to the SV40 promoter—a strong viral promoter derived from the Simian Virus 40 that is commonly used in molecular biology to drive high levels of gene expression in eukaryotic cells.

"Ivermectin inhibits importin alpha," McKernan notes. "Importin alpha is what drives that SV40 element, the enhancer and the promoter, into the nucleus. Alright? So it blocks that trafficking. And second to that the Fenben that he's [Makis] using has an impact on P53. It increases P53 activity. Alright?"

McKernan goes on to say:

"The other thing Fenben does is it's a beta tubulin disruptor. So it basically slows down microtubule formation. So cell cancer cells that are dividing very quickly have to make microtubules, which is like these ropes that rip the DNA apart and help in cell division. You may have seen pictures of cell division with spindles. Right? Those are micro tubules that are making that happen. And so if you have a quickly dividing cell, you have to be able to make microtubules very quickly. And beta tubulin is a protein involved in that.

"And Fenben actually inhibits that. So it slows the cell's ability to quickly divide and increases the P53 activity in the cell. So that's just what we need if this SV40 thing is, in fact, playing a role withP53 and if spike protein is, in fact, inhibiting P53. And then ivermectin just shuts down the ability to traffic that stuff into the nucleus. So it would shut off any avenue toward integration.

"So, the combo of those two drugs makes total sense to me for fighting anything that might be negatively associated with the Pfizer vaccine."
https://x.com/sensereceptor/status/1880287923087569332