8 months ago

185

http://www.DoctorsToTrust.com
presents episode 1080 | Dr Ken Sikaris
Low Carb Down Under podcast

Particles that end of in blood vessels, block them, has
NOTHING to do with nature...
LDL, instead of returning to liver, can stay in blood and
-become smaller, harder
-glycated or oxidized
Then CANNOT RETURN
TO LIVER
Rubbish pits collect it
[macrophages]
-if in blood vessels:
TROUBLE

Fructose can be converted to triglycerides [TG] in liver...
-normally sent out to tissues as VLDL fat packet
-if too much and no tissue wants it, it hangs around in blood'
'This is why you have high
TG: too much in blood and
all tissues are consuming
CARBS"
-VLDL starts crashing into
other particles
FATTY LIVER
[predicted by high blood TG]

VLDL correlates strongly with TG
-if high TG, high VLDL
How is VLDL level calculated?
Example
If TC 6
[LDL 4; HDL 1]
How get from TG to 6 TC?
Formula:
VLDL=TG/2.2
[no evidence for this formula]
-there is no other way
-When TG too high: Can't use formula

VLDL, high carb eaters make a lot of it...
-HDL trying to help out...CETP protein also helping
HDL is giving up its cholesterol ester & taking in VLDL fat
-eventually HDL disappears
in its effort to help VLDL
So
If TG goes UP: HDL DOWN
and
If TG goes DOWN: HDL UP

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doctorstotrust.com
lowcarbdownunder.com.au

Original Youtube: https://youtu.be/MXkE8_NdAyQ?si=ce1CsJOGFZGfglIG
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