2022 Research: A 16 Year Younger Biological Age; What Type of Walking?🚶🚶

A new study out of the University of Leicester has shown a link between the speed at which we walk and the speed at which our telomeres get shorter.

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https://www.nature.com/articles/s42003-022-03323-x
https://pubmed.ncbi.nlm.nih.gov/35444173/
https://www.researchgate.net/publication/360078941_Investigation_of_a_UK_biobank_cohort_reveals_causal_associations_of_self-reported_walking_pace_with_telomere_length
https://www.news-medical.net/news/20220505/Scientists-investigate-the-association-between-walking-pace-and-leucocyte-telomere-length.aspx

A Leicester-based team of researchers confirmed a causal link between walking pace and leucocyte telomere length (LTL), an indicator of biological age. The researchers estimated that a lifetime of brisk walking could lead to the equivalent of 16 years younger biological age by midlife. “Causal Associations of Self-Reported Walking Pace with Telomere Length” was published in Communications Biology. Whilst at the National Institute for Health Research (NIHR) Leicester Biomedical Research Center, Researchers from the University of Leicester studied genetic data from 405,981 middle-aged UK Biobank participants, they found that a faster walking pace, independent of the amount of physical activity, was associated with longer telomeres. 
For those that don’t know, Telomeres are the "caps" at the end of each chromosome, they hold repetitive sequences of non-coding DNA that protect the chromosome from damage. They perform much the same function as an aglet; an aglet is the small piece of plastic or metal at the end of a shoelace that stops it from unraveling.
Each time a cell divides, these telomeres become shorter, until they become so short that the cell can no longer divide, this is known as "replicative senescence." For this reason scientists consider LTL a strong marker for "biological age," independent from when an individual was born.
Although the relationship between telomere length and disease is not fully understood, the build-up of these senescent cells is believed to contribute to a range of symptoms we associate with aging, such as frailty and age-related diseases. While the physical, mental, health and social benefits of walking are very well-documented, this study is one - of the first of its kind - to compare genetic data with self-reported walking speeds
As well as actual measurements of movement intensity, from activity tracking devices worn by the participants.
Dr. Paddy Dempsey, a Lecturer and Research Fellow at the University of Leicester, and lead author said "Previous research on associations between walking pace, physical activity and telomere length has been limited by inconsistent findings and a lack of high-quality data. This research uses genetic data to provide stronger evidence for a causal link between faster walking pace and longer telomere length. Data from wrist-worn wearable activity tracking devices used to measure habitual physical activity also supported a stronger role of habitual activity intensity (e.g. faster walking) in relation to telomere length. This suggests measures such as a habitually slower walking speed are a simple way of identifying people at greater risk of chronic disease or unhealthy aging.
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