Epstein Borreliosis AIDS infinite antigen variation adapts to host DNA

ALL disease is Epstein Borreliosis AIDS infinite antigen variation adapts to host DNA, activated by any immune response...all the purdy ribbons of the rainbow: AIDS, Cancer, ALS, MS, Autism, Diabetes, Alzheimers, Morgellons etc .
In it's molecular forms called Prion Proteins
In it's most basic forms it is called Myco PLASMA, Ana PLASMOSIS, Toxo PLASMOSIS or Spiro PLASMA--Exosomes, Blebs, Gemma, TAU. And your baaad cholesterol.
In it's viral forms it is host gene dependent LEGIONS.
In its bacterial forms it is MANY esp. Ehrlichia and H.pylori, TB, According to where it resides .
It's infectious organisms protected in fungus. You can kill the infections but the immune suppression they cause isn't easily fixed.
All of your so called viruses are simply morphological forms of infinite host gene variations.....Their cystic viral morphologies and secreted blebs containing seed formerly known as Prion and flagellum that recombines and grows with infectious DNA.

RepA-WH1, the agent of an amyloid proteinopathy in bacteria, builds oligomeric pores through lipid vesicles https://pubmed.ncbi.nlm.nih.gov/26984374/
Possible roles of amyloids in malaria pathophysiology https://pubmed.ncbi.nlm.nih.gov/28031872/
Spiroplasma spp. biofilm formation is instrumental for their role in the pathogenesis of plant, insect and animal diseases https://pubmed.ncbi.nlm.nih.gov/22552100/
(RepA-WH1 -first synthetic bacterial prionoid)
Discussion
The results reported here point to a possible primary target for the cytotoxicity expressed by the RepA-WH1 prionoid in E. coli: the bacterial internal cell membrane. Three distinct mechanisms have been proposed for membrane disruption by amyloidogenic proteins8, including: i) the formation of pores by the assembly of protein monomers into ring-shaped oligomers; ii) membrane thinning and iii) lipid (detergent-like) extraction. Since the last mechanism should imply the lysis of a significant fraction of the vesicles, which was not the case under our experimental conditions, and the second one seems incompatible with the observed persistence of aggregates bound to the membranes (Figs 3–5), the formation of pores by protein insertion into the lipid bilayer, as first proposed for Aβ and α-synuclein39, was the most likely mechanism for vesicle leakage induced by RepA-WH1.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4794723/pdf/srep23144.pdf

https://www.lymeneteurope.org/forum/viewtopic.php?f=13&t=5399&hilit=anthrax&sid=34adf8c1b8ca65ea2c267f521d9994f1&start=10&fbclid=IwAR3EAz8iRggx4cM2Na4-tFppLL_Mpt6IUnjhhV5YrMD27BhG9m9bSIp15lE

https://www.lymeneteurope.org/forum/viewtopic.php?f=13&t=5147&start=30&fbclid=IwAR287sNwR32lfh8yXyEet7v-QtwDhwhK8BgXrNVaLCigh3_72fVn1Ylg95w

https://www.lymeneteurope.org/forum/viewtopic.php?f=6&t=4840&p=36246&fbclid=IwAR2y0lar0Z7OelH2bx99gVXNHABKbpFGWAjJaY9fso0IgTXy_qsXSaVo-8k#p36246

https://www.chicagotribune.com/news/breaking/chi-great-lakes-plastic-fibers-scientists-20150109-story.html

malaria epidemic 406,000 in 2018 http://hisz.rsoe.hu/alertmap/database/?pageid=event_summary&edis_id=EH-20180424-62784-VEN&fbclid=IwAR0SYJ-kxq44vPHLRmURj6b_iTy23q2DOqyPYXJPO0IgzZLCAVnr6yMrEYA

Treating Autism With Antibiotics (France 3 19/20, Feb 17, 2012)
https://www.youtube.com/watch?v=yOno_2m_8LY&fbclid=IwAR3KBiW_h8C2okAY7U-PQNO2T4f4bTc1ROLvSzKjdA5l3KmGBbDVmj8XFok&app=desktop

Artemisinin and its derivatives in treating protozoan infections beyond malaria
https://pubmed.ncbi.nlm.nih.gov/27867026/

Good pics of Spirochetal AIDS. Notice the round bodies still there.
https://pdfs.semanticscholar.org/017c/f5dacaf19d207de7f5e51eb75dd847e20446.pdf

https://sites.google.com/view/doxy-cancer/home?fbclid=IwAR1wovyL3Y7g7UAjvyr1gO7ul10ohQRMnsdtsWtzz7faksu_A0B0ZRqsNO4